J. Richards

Antioxidants in bronchoalveolar lavage fluid cells isolated from ozone--exposed normal and ascorbate-deficient guinea pigs.

U. Kodavanti, D. Costa, G. Hatch, 1996, Experimental lung research.

The spontaneously hypertensive rat as a model of human cardiovascular disease: evidence of exacerbated cardiopulmonary injury and oxidative stress from inhaled emission particulate matter.

A. Ledbetter, M. Schladweiler, U. Kodavanti, 2000, Toxicology and applied pharmacology.

Lung injury from intratracheal and inhalation exposures to residual oil fly ash in a rat model of monocrotaline-induced pulmonary hypertension.

A. Ledbetter, U. Kodavanti, D. Costa, 1999, Journal of toxicology and environmental health. Part A.

Acute lung injury from intratracheal exposure to fugitive residual oil fly ash and its constituent metals in normo- and spontaneously hypertensive rats.

M. Schladweiler, U. Kodavanti, D. Costa, 2001, Inhalation toxicology.

Oxidative interactions of synthetic lung epithelial lining fluid with metal-containing particulate matter.

G. Hatch, R. Slade, K. Crissman, 2001, American journal of physiology. Lung cellular and molecular physiology.

TEMPORAL ASSOCIATION BETWEEN PULMONARY AND SYSTEMIC EFFECTS OF PARTICULATE MATTER IN HEALTHY AND CARDIOVASCULAR COMPROMISED RATS

David C Christiani, Daniel L Costa, Russ Hauser, 2002, Journal of toxicology and environmental health. Part A.