A. Jox

发表

Detection of identical Hodgkin-Reed Sternberg cell specific immunoglobulin gene rearrangements in a patient with Hodgkin's disease of mixed cellularity subtype at primary diagnosis and in relapse two and a half years later.

V. Diehl, T. Zander, R. Küppers, 1998, Annals of oncology : official journal of the European Society for Medical Oncology.

Hodgkin's disease - new treatment strategies toward the cure of patients.

V. Diehl, M. Sieber, A. Jox, 1999, Cancer treatment reviews.

Isolation of viable Hodgkin and Reed-Sternberg cells from Hodgkin disease tissues.

V. Diehl, K. Rajewsky, H. Tesch, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Clonal relapse in Hodgkin's disease.

V. Diehl, T. Zander, A. Jox, 1997, The New England journal of medicine.

Suppression of Burkitt's lymphoma tumorigenicity in nude mice by co‐inoculation of EBV‐immortalized lymphoblastoid cells

M. Pawlita, P. Möller, H. Bohlen, 1995, International journal of cancer.

HMGI-C, a member of the high mobility group family of proteins, is expressed in hematopoietic stem cells and in leukemic cells.

P. Rogalla, J. Bullerdiek, C. Kalle, 1997, Leukemia & lymphoma.

Somatic Mutations Within the Untranslated Regions of Rearranged Ig Genes in a Case of Classical Hodgkin’s Disease as a Potential Cause for the Absence of Ig in the Lymphoma Cells

V. Diehl, T. Zander, R. Küppers, 1999 .

Morphologically normal, CD30‐negative B‐lymphocytes with chromosome aberrations in classical Hodgkin's disease: the progenitor cell of the malignant clone?

J. Arends, M. Stevens-Kroef, A. Hopman, 1999, The Journal of pathology.

Somatic mutations within the untranslated regions of rearranged Ig genes in a case of classical Hodgkin's disease as a potential cause for the absence of Ig in the lymphoma cells.

V. Diehl, T. Zander, R. Küppers, 1999, Blood.

Hodgkin's disease biology: recent advances

V. Diehl, A. Jox, J. Wolf, 1997, Hematological oncology.

Selective loss of integrated Epstein-Barr virus genomes after long-term cultivation of Burkitt's lymphoma x B-lymphoblastoid cell hybrids due to chromatin instability at the integration site.

V. Diehl, M. Pawlita, J. Bullerdiek, 1995, Virology.

Detection of a Hodgkin/Reed‐Sternberg cell specific immunoglobulin gene rearrangement in the serum DNA of a patient with Hodgkin's disease

V. Diehl, H. Tesch, H. Bohlen, 1999, British journal of haematology.

Stable nontumorigenic phenotype of somatic cell hybrids between malignant Burkitt's lymphoma cells and autologous EBV-immortalized B cells despite induction of chromosomal breakage and loss.

V. Diehl, M. Pawlita, P. Möller, 1998, Cancer research.

Integration of Epstein Barr virus near the breakpoint of a translocation 11;19 in a Burkitt's lymphoma cell line.

V. Diehl, M. Pawlita, J. Bullerdiek, 1993, Cancer genetics and cytogenetics.

Integration of Epstein-Barr virus in Burkitt's lymphoma cells leads to a region of enhanced chromosome instability.

V. Diehl, M. Pawlita, J. Bullerdiek, 1997, Annals of oncology : official journal of the European Society for Medical Oncology.

The biology of Hodgkin's disease

V. Diehl, A. Jox, J. Wolf, 1997 .

The nature of Reed-Sternberg cells, lymphocytic and histiocytic cells and their molecular biology in Hodgkin’s disease.

H. Stein, T. Marafioti, M. Hummel, 1999 .