S. Turban

发表

Transcriptional effect of hypoxia on placental leptin

S. Hauguel-de Mouzon, J. Challier, M. Guerre-Millo, 2001, FEBS letters.

Protein kinase C isoforms: Mediators of reactive lipid metabolites in the development of insulin resistance

É. Hajduch, S. Turban, 2011, FEBS letters.

Adipose tissue-specific increase in angiotensinogen expression and secretion in the obese (fa/fa) Zucker rat.

P. Ferré, A. Quignard-Boulangé, I. Hainault, 2002, American journal of physiology. Endocrinology and metabolism.

Intracellular ceramide synthesis and protein kinase Czeta activation play an essential role in palmitate-induced insulin resistance in rat L6 skeletal muscle cells.

É. Hajduch, H. S. Hundal, A. Gray, 2004, The Biochemical journal.

PHOSPHO1 is a skeletal regulator of insulin resistance and obesity

J. Millán, K. Suchacki, C. Farquharson, 2020, BMC biology.

MG 53 is not a critical regulator of insulin signaling pathway in 3 skeletal muscle 4 5 6

P. Delerive, C. Philouze, N. Provost, 2021 .

MG53 is not a critical regulator of insulin signaling pathway in skeletal muscle.

P. Delerive, C. Philouze, N. Provost, 2021, PloS one.

MG53 is not a critical regulator of insulin signaling pathway in skeletal muscle

P. Delerive, C. Philouze, N. Provost, 2020, bioRxiv.

Molecular and cellular mechanisms of adipose secretion: Comparison of leptin and angiotensinogen

P. Ferré, M. Guerre-Millo, I. Hainault, 2001, Journal of cellular biochemistry.

Insulin-stimulated glucose uptake does not require p38 mitogen-activated protein kinase in adipose tissue or skeletal muscle.

J. Arthur, K. Sakamoto, É. Hajduch, 2005, Diabetes.

Increased Angiogenesis Protects against Adipose Hypoxia and Fibrosis in Metabolic Disease-resistant 11β-Hydroxysteroid Dehydrogenase Type 1 (HSD1)-deficient Mice*

B. Walker, J. Iredale, P. Ratcliffe, 2011, The Journal of Biological Chemistry.

Hypoxia increases leptin expression in human PAZ6 adipose cells

T. Issad, V. Zilberfarb, M. Guerre-Millo, 2002, Diabetologia.

Targeting of PKCzeta and PKB to caveolin-enriched microdomains represents a crucial step underpinning the disruption in PKB-directed signalling by ceramide.

N. Dimopoulos, É. Hajduch, H. S. Hundal, 2008, The Biochemical journal.

Embryonic expression of the leptin receptor gene in mesoderm-derived tissues.

M. Abitbol, M. Guerre-Millo, S. Turban, 2002, Comptes rendus biologies.

Specific increase in leptin production in obese (falfa) rat adipose cells.

P. Ferré, M. Guerre-Millo, J. Truccolo, 2002, The Biochemical journal.

Angiotensinogen-deficient mice exhibit impairment of diet-induced weight gain with alteration in adipose tissue development and increased locomotor activity.

A. Fukamizu, A. Geloen, M. Teboul, 2001, Endocrinology.

Characterising the Inhibitory Actions of Ceramide upon Insulin Signaling in Different Skeletal Muscle Cell Models: A Mechanistic Insight

F. Foufelle, P. Ferré, É. Hajduch, 2014, PloS one.

Leptin receptor-deficient obese Zucker rats reduce their food intake in response to a systemic supply of calories from glucose.

C. Magnan, M. Guerre-Millo, M. Gilbert, 2003, Diabetes.

Novel Fat Depot–Specific Mechanisms Underlie Resistance to Visceral Obesity and Inflammation in 11β-Hydroxysteroid Dehydrogenase Type 1–Deficient Mice

J. Seckl, T. Kipari, J. Hughes, 2011, Diabetes.

Preadipocyte 11 -Hydroxysteroid Dehydrogenase Type 1 Is a Keto-Reductase and Contributes to Diet-Induced Visceral Obesity in Vivo

J. Seckl, N. Morton, N. Morton, 2008 .

Angiotensinogen-Deficient Mice Exhibit Impairment of Diet-Induced Weight Gain with Alteration in Adipose Tissue Development and Increased Locomotor Activity.

A. Fukamizu, A. Geloen, M. Teboul, 2001, Endocrinology.

Optimal Elevation of β-Cell 11β-Hydroxysteroid Dehydrogenase Type 1 Is a Compensatory Mechanism That Prevents High-Fat Diet–Induced β-Cell Failure

B. Walker, J. Seckl, J. Mullins, 2012, Diabetes.

Optimal Elevation of b -Cell 11 b -Hydroxysteroid Dehydrogenase Type 1 Is a Compensatory Mechanism that Prevents High Fat Diet – Induced b -Cell Failure

J. Seckl, J. Mullins, N. Morton, 2012 .

Insulin regulates the expression of the GLUT5 transporter in L6 skeletal muscle cells

É. Hajduch, H. S. Hundal, É. Brot-Laroche, 2003, FEBS letters.

β-Cell-Specific Glucocorticoid Reactivation Attenuates Inflammatory β-Cell Destruction

B. Walker, J. Seckl, N. Morton, 2014, Front. Endocrinol..

Pregnancy in Obese Mice Protects Selectively against Visceral Adiposity and Is Associated with Increased Adipocyte Estrogen Signalling

T. Kipari, P. Saunders, J. Norman, 2014, PloS one.

Defining the Contribution of AMP-activated Protein Kinase (AMPK) and Protein Kinase C (PKC) in Regulation of Glucose Uptake by Metformin in Skeletal Muscle Cells*

B. Viollet, A. Marette, D. Hardie, 2012, The Journal of Biological Chemistry.

Targeting of PKCzeta and PKB to caveolin-enriched microdomains represents a crucial step underpinning the disruption in PKB-directed signaling by ceramide

N. Dimopoulos, É. Hajduch, I. Dugail, 2021 .

Preadipocyte 11beta-hydroxysteroid dehydrogenase type 1 is a keto-reductase and contributes to diet-induced visceral obesity in vivo.

J. Seckl, N. Morton, K. Chapman, 2008, Endocrinology.

Protein kinase C isoforms: Mediators of reactive lipid metabolites in the development of insulin resistance

S. Turban, E. Hajduch, 2011, FEBS letters.