P. C. Viswanathan

发表

Mission Management Computer Software for RLV-TD

V. R. Lalithambika, Kesavabrahmaji Karuturi, A. Abdul Shukkoor, 2017 .

Two distinct congenital arrhythmias evoked by a multidysfunctional Na(+) channel.

P. C. Viswanathan, A. Wilde, J. Balser, 2000, Circulation research.

A sodium-channel mutation causes isolated cardiac conduction disease

P. C. Viswanathan, A. Wilde, H. Tan, 2001, Nature.

Effects of IKr and IKs heterogeneity on action potential duration and its rate dependence: a simulation study.

P. C. Viswanathan, Y. Rudy, R. Shaw, 1999, Circulation.

Genetics of acquired long QT syndrome.

P. C. Viswanathan, D. Roden, 2005, The Journal of clinical investigation.

Potential Role of Isoketals Formed Via the Isoprostane Pathway of Lipid Peroxidation in Ischemic Arrhythmias

P. C. Viswanathan, J. Balser, L. Roberts, 2007, Journal of cardiovascular pharmacology.

Pause induced early afterdepolarizations in the long QT syndrome: a simulation study.

Y Rudy, P. C. Viswanathan, Y. Rudy, 1999, Cardiovascular research.

A common SCN 5 A polymorphism modulates the biophysical effects of an SCN 5 A mutation

P. C. Viswanathan, J. Balser, D. Benson, 2003 .

Mutation in Glycerol-3-Phosphate Dehydrogenase 1–Like Gene (GPD1-L) Decreases Cardiac Na+ Current and Causes Inherited Arrhythmias

P. C. Viswanathan, M. Ackerman, R. Weiss, 2007, Circulation.

Quantitation of protein kinase A-mediated trafficking of cardiac sodium channels in living cells.

P. C. Viswanathan, K. Murray, K. Wells, 2006, Cardiovascular research.

Allelic Variants in Long-QT Disease Genes in Patients With Drug-Associated Torsades de Pointes

P. C. Viswanathan, S. Hohnloser, K. Murray, 2002, Circulation.

Sex, Age, and Regional Differences in L-Type Calcium Current Are Important Determinants of Arrhythmia Phenotype in Rabbit Hearts With Drug-Induced Long QT Type 2

P. C. Viswanathan, G. Salama, W. Walker, 2008, Circulation research.

Compound‐specific Na+ channel pore conformational changes induced by local anaesthetics

P. C. Viswanathan, J. Balser, T. Nakajima, 2005, The Journal of physiology.

Cellular arrhythmogenic effects of congenital and acquired long-QT syndrome in the heterogeneous myocardium.

Y Rudy, P. C. Viswanathan, Y. Rudy, 2000, Circulation.

Clinical, Genetic, and Biophysical Characterization of SCN5A Mutations Associated With Atrioventricular Conduction Block

P. C. Viswanathan, J. Balser, A. George, 2002, Circulation.

A common SCN5A polymorphism modulates the biophysical effects of an SCN5A mutation.

P. C. Viswanathan, J. Balser, D. Benson, 2003, The Journal of clinical investigation.

Molecular basis of isolated cardiac conduction disease.

P. C. Viswanathan, J. Balser, 2006, Handbook of experimental pharmacology.

Oxidative Mediated Lipid Peroxidation Recapitulates Proarrhythmic Effects on Cardiac Sodium Channels

Mark E. Anderson, P. C. Viswanathan, Jeffrey R. Balser, 2005, Circulation research.

A sodium channel pore mutation causing Brugada syndrome.

P. C. Viswanathan, V. Shusterman, R. Weiss, 2007, Heart rhythm.

Recreating an artificial biological pacemaker: insights from a theoretical model.

P. C. Viswanathan, Vinod Sharma, D. Sigg, 2006, Heart rhythm.

Inherited sodium channelopathies: a continuum of channel dysfunction.

P. C. Viswanathan, J. Balser, 2004, Trends in cardiovascular medicine.

SCN5A-linked disease syndromes: complex monogenic disorders of cardiac rhythm.

P. C. Viswanathan, 2005, Future cardiology.

New Mechanism Contributing to Drug-Induced Arrhythmia: Rescue of a Misprocessed LQT3 Mutant

P. C. Viswanathan, D. Roden, Tao Yang, 2005, Circulation.

LQT 3 Mutant New Mechanism Contributing to Drug-Induced Arrhythmia : Rescue of a Misprocessed

P. C. Viswanathan, D. Roden, Tao Yang, 2005 .

Functional Interactions between Distinct Sodium Channel Cytoplasmic Domains through the Action of Calmodulin*

P. C. Viswanathan, J. Balser, W. Chazin, 2009, Journal of Biological Chemistry.

Gating-Dependent Mechanisms for Flecainide Action in SCN5A-Linked Arrhythmia Syndromes

P. C. Viswanathan, D. Roden, J. Balser, 2001, Circulation.

HERG Is Protected from Pharmacological Block by α-1,2-Glucosyltransferase Function*

P. C. Viswanathan, J. Balser, S. Kupershmidt, 2007, Journal of Biological Chemistry.

HERG is protected from pharmacological block by alpha-1,2-glucosyltransferase function.

P. C. Viswanathan, J. Balser, S. Kupershmidt, 2007, The Journal of biological chemistry.

Novel characteristics of a misprocessed mutant HERG channel linked to hereditary long QT syndrome.

P. C. Viswanathan, E. Kaufman, S. Priori, 2000, American journal of physiology. Heart and circulatory physiology.