J. Volquardsen

Impaired 53BP1/RIF1 DSB mediated end-protection stimulates CtIP-dependent end resection and switches the repair to PARP1-dependent end joining in G1

K. Rothkamm, E. Dikomey, C. Petersen, 2016, Oncotarget.

Functional crosstalk between DNA damage response proteins 53BP1 and BRCA1 regulates double strand break repair choice.

K. Rothkamm, R. Reimer, E. Dikomey, 2016, Radiotherapy and oncology : journal of the European Society for Therapeutic Radiology and Oncology.

Loss of PTEN-assisted G2/M checkpoint impedes homologous recombination repair and enhances radio-curability and PARP inhibitor treatment response in prostate cancer

R. Simon, M. Kluth, C. Hube-Magg, 2018, Scientific Reports.

Fully automated counting of DNA damage foci in tumor cell culture: A matter of cell separation.

K. Rothkamm, C. Petersen, W. Mansour, 2021, DNA repair.

Loss of PTEN-assisted G2/M checkpoint impedes homologous recombination repair and enhances radio-curability and PARP inhibitor treatment response in prostate cancer

R. Simon, M. Kluth, C. Hube-Magg, 2018, Scientific Reports.

A functional ex vivo assay to detect PARP1‐EJ repair and radiosensitization by PARP‐inhibitor in prostate cancer

T. Schlomm, B. Beyer, T. Lange, 2019, International journal of cancer.

BCL2-overexpressing prostate cancer cells rely on PARP1-dependent end-joining and are sensitive to combined PARP inhibitor and radiation therapy.

C. Bokemeyer, R. Simon, K. Rothkamm, 2018, Cancer letters.