L. Deonarine

发表

CHD1 Loss Alters AR Binding at Lineage-Specific Enhancers and Modulates Distinct Transcriptional Programs to Drive Prostate Tumorigenesis.

M. Rubin, O. Elemento, Noah Dephoure, 2019, Cancer cell.

SPOP mutation drives prostate neoplasia without stabilizing oncogenic transcription factor ERG

Jonathan E. Shoag, M. Rubin, A. Sboner, 2017, The Journal of clinical investigation.

SPOP Mutation Drives Prostate Tumorigenesis In Vivo through Coordinate Regulation of PI3K/mTOR and AR Signaling.

M. Rubin, A. Chinnaiyan, A. Sboner, 2017, Cancer cell.

G3BP1 inhibits Cul3SPOP to amplify AR signaling and promote prostate cancer

M. Rubin, A. Chinnaiyan, A. Sboner, 2021, Nature Communications.

SPOP mutation confers sensitivity to AR-targeted therapy in prostate cancer by reshaping the androgen-driven chromatin landscape

O. Elemento, A. Sboner, E. Davicioni, 2021, bioRxiv.

Patient derived organoids to model rare prostate cancer phenotypes

Dong Gao, Matteo Benelli, Davide Prandi, 2018, Nature Communications.

Reshaping of the androgen-driven chromatin landscape in normal prostate cells by early cancer drivers and effect on therapeutic sensitivity

O. Elemento, A. Sboner, E. Davicioni, 2021, Cell reports.

CHD1 Loss Alters AR Binding at Lineage-Specific Enhancers and Modulates Distinct Transcriptional Programs to Drive Prostate Tumorigenesis.

M. Rubin, O. Elemento, Noah Dephoure, 2019, Cancer cell.

The role of photovaporization of the prostate in small volume benign prostatic hyperplasia and review of the literature

K. Zorn, V. Misrai, B. Chughtai, 2019, Asian journal of urology.

Patient derived organoids to model rare prostate cancer phenotypes

M. Rubin, O. Elemento, A. Sboner, 2018, Nature Communications.

Canonical AREs are tumor suppressive regulatory elements in the prostate

A. Sboner, E. Davicioni, F. Khani, 2024, bioRxiv.