K. C. Mattos

Exercise training reduces cardiac angiotensin II levels and prevents cardiac dysfunction in a genetic model of sympathetic hyperactivity-induced heart failure in mice

J. Krieger, M. Irigoyen, E. Oliveira, 2009, European Journal of Applied Physiology.

Lack of β2‐AR improves exercise capacity and skeletal muscle oxidative phenotype in mice

P. Brum, A. V. Bacurau, C. R. Bueno Júnior, 2012, Scandinavian journal of medicine & science in sports.

Intracellular mechanisms of specific beta-adrenoceptor antagonists involved in improved cardiac function and survival in a genetic model of heart failure.

J. Krieger, S. Guatimosim, L. Tanaka, 2008, Journal of molecular and cellular cardiology.

Exercise training improves the net balance of cardiac Ca2+ handling protein expression in heart failure.

J. Krieger, M. Irigoyen, C. Negrão, 2007, Physiological genomics.

Angiotensin receptor blockade improves the net balance of cardiac Ca(2+) handling-related proteins in sympathetic hyperactivity-induced heart failure.

P. Brum, J. B. Moreira, J. C. Ferreira, 2011, Life sciences.

Exercise training delays cardiac dysfunction and prevents calcium handling abnormalities in sympathetic hyperactivity-induced heart failure mice.

J. Krieger, M. Irigoyen, C. Negrão, 2008, Journal of applied physiology.

Lack of β2-adrenoceptors aggravates heart failure-induced skeletal muscle myopathy in mice

C. Negrão, D. Casarini, P. Brum, 2014, Journal of cellular and molecular medicine.

Swimming Physical Training Promotes Cardiac Remodeling and Improves Blood Perfusion in the Cardiac Muscle of SHR Via Adenosine-Dependent Mechanism

J. G. Barros, M. Irigoyen, E. Oliveira, 2011 .

Exercise training improves aortic depressor nerve sensitivity in rats with ischemia-induced heart failure.

H. Middlekauff, É. Moreira, M. Irigoyen, 2006, American journal of physiology. Heart and circulatory physiology.