H. Wick

发表

Acute cortisol excess results in unimpaired insulin action on lipolysis and branched chain amino acids, but not on glucose kinetics and C-peptide concentrations in man.

U. Keller, D. Clerc, H. Wick, 1986, Metabolism: clinical and experimental.

Analysis of glycogen storage disease by in vivo 13C NMR comparison of normal volunteers with a patient

J. Seelig, N. Beckmann, H. Wick, 1990, Magnetic resonance in medicine.

Comparison of patients with complete and partial biotinidase deficiency: Biochemical studies

T. Suormala, E. R. Baumgartner, S. Schweitzer, 1990, Journal of Inherited Metabolic Disease.

Five Patients with a Biotin-Responsive Defect in Holocarboxylase Formation: Evaluation of Responsiveness to Biotin Therapy in Vivo and Comparative Biochemical Studies in Vi1

T. Suormala, M. Durán, A. Fuchshuber, 1997, Pediatric Research.

Intestinal absorption and renal excretion of biotin in patients with biotinidase deficiency

T. Suormala, E. R. Baumgartner, H. Wick, 1985, European Journal of Pediatrics.

Biotinidase Deficiency Associated with Renal Loss of Biocytin and Biotin

T. Suormala, E. R. Baumgartner, H. Wick, 1985, Annals of the New York Academy of Sciences.

Quantitative determination of biocytin in urine of patients with biotinidase deficiency using high-performance liquid chromatography (HPLC).

T. Suormala, E. R. Baumgartner, J. Bausch, 1988, Clinica chimica acta; international journal of clinical chemistry.

Infantile multiple carboxylase deficiency: evidence for normal intestinal absorption but renal loss of biotin.

T. Suormala, R. Baumgartner, J. Geisert, 1982, Helvetica paediatrica acta.

Experience with insulin-induced hypoglycemia as a provocative test for growth hormone and cortisol secretion in children.

J. Girard, J. Baumann, H. Wick, 1971, Hormones.

An improved method for preparation of samples for the simultaneous assay of some antiepileptic drugs by gas-liquid chromatography.

H. Wick, K. Schweizer, T. Brechbühler, 1978, Clinica chimica acta; international journal of clinical chemistry.

Late-onset holocarboxylase synthetase-deficiency: pre- and post-natal diagnosis and evaluation of effectiveness of antenatal biotin therapy

T. Suormala, E. R. Baumgartner, M. Durán, 1998, European Journal of Pediatrics.

Biotinidase Deficiency: A Cause of Subacute Necrotizing Encephalomyelopathy (Leigh Syndrome). Report of a Case with Lethal Outcome

T. Suormala, E. R. Baumgartner, A. Probst, 1989, Pediatric Research.

Low biotinidase activity in plasma of some preterm infants: possible source of false-positive screening results

E. R. Baumgartner, H. Wick, T. Sourmala, 1988, European Journal of Pediatrics.

Biotinidase deficiency: Factors responsible for the increased biotin requirement

T. Suormala, E. R. Baumgartner, J. Bausch, 1985, Journal of Inherited Metabolic Disease.

Na(+)-dependent biotin transport into brush-border membrane vesicles from rat kidney.

E. R. Baumgartner, H. Wick, B. Baur, 1990, The American journal of physiology.

Rapid differential diagnosis of carboxylase deficiencies and evaluation for biotin-responsiveness in a single blood sample.

T. Suormala, E. R. Baumgartner, H. Wick, 1985, Clinica chimica acta; international journal of clinical chemistry.

Biotin-responsive multiple carboxylase deficiency (MCD): deficient biotinidase activity associated with renal loss of biotin.

T. Suormala, E. R. Baumgartner, H. Wick, 1984, Journal of inherited metabolic disease.

Variants of citrullinaemia.

M. Mihatsch, C. Bachmann, J. Colombo, 1973, Archives of disease in childhood.

Comparison of Folic Acid Coenzyme Distribution Patterns in Patients with Methylenetetrahydrofolate Reductase and Methionine Synthetase Deficiencies

E. R. Baumgartner, E. Stokstad, H. Wick, 1985, Pediatric Research.

Congenital defect in intracellular cobalamin metabolism resulting in homocysteinuria and methylmalonic aciduria. I. Case report and histopathology.

E. R. Baumgartner, B. Steinmann, R. Maurer, 1979, Helvetica paediatrica acta.

Congenital defect in intracellular cobalamin metabolism resulting in homocystinuria and methylmalonic aciduria. II. Biochemical investigations.

E. R. Baumgartner, J. Linnell, C. Bachmann, 1979, Helvetica paediatrica acta.

Thiamine dependency in a patient with congenital lacticacidaemia due to pyruvate dehydrogenase deficiency

R. Baumgartner, H. Wick, K. Schweizer, 1977, Agents and Actions.

Foamy myocardial transformation in a child with a disturbed respiratory chain

R. Sengers, U. Ketelsen, W. Ruitenbeek, 1987, European Journal of Pediatrics.

Vascular lesions in two patients with congenital homocystinuria due to different defects of remethylation

A. Probst, R. Maurer, R. Baumgartner, 1980, Journal of Inherited Metabolic Disease.

Acute Neonatal Nonketotic Hyperglycinemia: Normal Propionate and Methylmalonate Metabolism

E. R. Baumgartner, C. Bachmann, H. Wick, 1975, Pediatric Research.

[Normal propionate metabolism in "non-ketotic hyperglycinemia"].

E. R. Baumgartner, H. Wick, 1972, The New England journal of medicine.

Abnormal cerebral hemodynamics during attacks of alternating hemiplegia

H. Wick, H. Bode, 1992, Child's Nervous System.

Homozygous hypobetalipoproteinaemia and phenylketonuria

W. Tackmann, J. Leititis, H. Wick, 1985, European Journal of Pediatrics.

Liver morphology in a case of citrullinemia (a light and electron microscopic study).

M. Mihatsch, C. Bachmann, U. Riede, 1974, Beitrage zur Pathologie.

[Non-ketotic hyperglycinemia: peracute course in neonatal period].

M. Mihatsch, C. Bachmann, A. Olafsson, 1971, Helvetica paediatrica acta.