Kota Ishikawa

发表

Ingestion of a moderate high‐sucrose diet results in glucose intolerance with reduced liver glucokinase activity and impaired glucagon‐like peptide‐1 secretion

N. Ozaki, Y. Oiso, N. Inagaki, 2012, Journal of diabetes investigation.

Secreted factors from dental pulp stem cells improve glucose intolerance in streptozotocin-induced diabetic mice by increasing pancreatic β-cell function

H. Arima, Y. Oiso, Shinichi Tsunekawa, 2015, BMJ Open Diabetes Research and Care.

Central diabetes insipidus and hypothalamic hypothyroidism associated with aceruloplasminemia.

Minemori Watanabe, H. Miyajima, S. Kono, 2010, Internal medicine.

Long-Term Pancreatic Beta Cell Exposure to High Levels of Glucose but Not Palmitate Induces DNA Methylation within the Insulin Gene Promoter and Represses Transcriptional Activity

N. Ozaki, Y. Kondo, Y. Sugimura, 2015, PloS one.

Urine output and resultant osmotic water shift are major determinants of plasma sodium level in syndrome of inappropriate antidiuretic hormone secretion.

Minemori Watanabe, Y. Oiso, T. Tsunekawa, 2013, Translational research : the journal of laboratory and clinical medicine.

KATP channel as well as SGLT1 participates in GIP secretion in the diabetic state.

N. Ozaki, S. Seino, Y. Hayashi, 2014, The Journal of endocrinology.

Effect of hyperglycemia on hepatocellular carcinoma development in diabetes.

H. Goto, T. Honda, M. Ishigami, 2015, Biochemical and biophysical research communications.

Endogenous GIP ameliorates impairment of insulin secretion in proglucagon-deficient mice under moderate beta cell damage induced by streptozotocin

N. Ozaki, Y. Hayashi, H. Arima, 2016, Diabetologia.

Endogenous GIP ameliorates impairment of insulin secretion in proglucagon-deficient mice under moderate beta cell damage induced by streptozotocin

N. Ozaki, Y. Hayashi, H. Arima, 2016, Diabetologia.

KATP channel as well as SGLT1 participates in GIP secretion in the diabetic state.

N. Ozaki, S. Seino, Y. Hayashi, 2014, The Journal of endocrinology.

A novel mechanism of idiopathic orthostatic hypotension and hypocatecholaminemia due to autoimmunity against aromatic l-Amino acid decarboxylase.

Y. Sugimura, Shinichi Tsunekawa, Y. Seino, 2024, Biochemical and biophysical research communications.