I. Koziollek-Drechsler

Wild-type Cu/Zn superoxide dismutase stabilizes mutant variants by heterodimerization

N. Hellmann, A. Chakrabartty, H. Decker, 2014, Neurobiology of Disease.

Wild-type Cu/Zn superoxide dismutase (SOD1) does not facilitate, but impedes the formation of protein aggregates of amyotrophic lateral sclerosis causing mutant SOD1

H. Neumann, C. Behl, A. Clement, 2009, Neurobiology of Disease.

Dimerization of visinin-like protein 1 is regulated by oxidative stress and calcium and is a pathological hallmark of amyotrophic lateral sclerosis.

S. Tenzer, H. Schild, C. Behl, 2014, Free radical biology & medicine.

Heterodimer formation of wild-type and amyotrophic lateral sclerosis-causing mutant Cu/Zn-superoxide dismutase induces toxicity independent of protein aggregation.

C. Behl, R. Wade, A. Clement, 2008, Human molecular genetics.

Receptor-mediated endocytosis 8 (RME-8)/DNAJC13 is a novel positive modulator of autophagy and stabilizes cellular protein homeostasis

C. Freese, Luise Florin, C. Behl, 2020, Cellular and Molecular Life Sciences.

Receptor-mediated endocytosis 8 (RME-8)/DNAJC13 is a novel positive modulator of autophagy and stabilizes cellular protein homeostasis

C. Freese, Luise Florin, C. Behl, 2020, Cellular and Molecular Life Sciences.

Estrogen receptor α regulates non-canonical autophagy that provides stress resistance to neuroblastoma and breast cancer cells and involves BAG3 function

U. Wolfrum, D. Kögel, C. Behl, 2015, Cell Death and Disease.

RAB3GAP1 and RAB3GAP2 modulate basal and rapamycin-induced autophagy

C. Behrends, C. Behl, Jennifer Jung, 2014, Autophagy.