C. F. Lee

发表

Failed Power Plant Turns Into Mass Murder: New Insight on Mitochondrial Cardiomyopathy.

R. Tian, Yang Cao, C. F. Lee, 2018, Circulation research.

NAD+ Redox Imbalance in the Heart Exacerbates Diabetic Cardiomyopathy

Y. Chiao, J. Sadoshima, R. Tian, 2020, bioRxiv.

Elimination of NADPH Oxidase Activity Promotes Reductive Stress and Sensitizes the Heart to Ischemic Injury

J. Sadoshima, R. Tian, Wang Wang, 2014, Journal of the American Heart Association.

Nox4 is a Novel Inducible Source of Reactive Oxygen Species in Monocytes and Macrophages and Mediates Oxidized Low Density Lipoprotein–Induced Macrophage Death

K. Schröder, Q. Zhao, R. Asmis, 2010, Circulation research.

Normalization of NAD+ Redox Balance as a Therapy for Heart Failure

D. Goodlett, Y. Chiao, Y. Goo, 2016, Circulation.

Genetic architecture of heart mitochondrial proteome influencing cardiac hypertrophy

David E. James, M. Pellegrini, A. Lusis, 2021, bioRxiv.

Harnessing NAD+ Metabolism as Therapy for Cardiometabolic Diseases

Y. Chiao, C. F. Lee, A. Chakraborty, 2022, Current Heart Failure Reports.

BIGH3 protein and macrophages in retinal endothelial cell apoptosis

A. Tsin, R. LeBaron, H. Kim, 2014, Apoptosis.

Targeting NAD+ Metabolism as Interventions for Mitochondrial Disease

R. Tian, G. A. Nagana Gowda, G. Gowda, 2019, Scientific Reports.

Simultaneous Analysis of Major Coenzymes of Cellular Redox Reactions and Energy Using ex Vivo 1H NMR Spectroscopy

R. Tian, D. Raftery, G. A. Nagana Gowda, 2016, Analytical chemistry.

Mitochondrial complex I deficiency increases protein acetylation and accelerates heart failure.

Wang Wang, Georgios Karamanlidis, Lorena Garcia-Menendez, 2013, Cell metabolism.

Thiol Oxidative Stress Induced by Metabolic Disorders Amplifies Macrophage Chemotactic Responses and Accelerates Atherogenesis and Kidney Injury in LDL Receptor–Deficient Mice

C. Phelix, R. LeBaron, Lisa R Tannock, 2009, Arteriosclerosis, thrombosis, and vascular biology.

Promoting PGC-1α-driven mitochondrial biogenesis is detrimental in pressure-overloaded mouse hearts.

R. Tian, Stephen C. Kolwicz, G. Karamanlidis, 2014, American journal of physiology. Heart and circulatory physiology.

Redox regulation of MAPK phosphatase 1 controls monocyte migration and macrophage recruitment

H. Kim, S. Ullevig, D. Zamora, 2012, Proceedings of the National Academy of Sciences.

Revealing Pathway Dynamics in Heart Diseases by Analyzing Multiple Differential Networks

Peng Gao, Rong Tian, Long Gao, 2015, PLoS Comput. Biol..

NADPH Oxidase 4 Mediates Monocyte Priming and Accelerated Chemotaxis Induced by Metabolic Stress

S. Ullevig, Q. Zhao, C. Lee, 2012, Arteriosclerosis, thrombosis, and vascular biology.

Chemical Crosslinking Mass Spectrometry Analysis of Protein Conformations and Supercomplexes in Heart Tissue.

Andrew Keller, Juan D Chavez, James E Bruce, 2017, Cell systems.

Mitochondrial protein interactome elucidated by chemical cross-linking mass spectrometry

Juan D. Chavez, James E. Bruce, Devin K. Schweppe, 2017, Proceedings of the National Academy of Sciences.

Targeting NAD+ Metabolism as Interventions for Mitochondrial Disease

R. Tian, G. A. Nagana Gowda, C. F. Lee, 2019, Scientific Reports.

Regulation of Monocyte Adhesion and Migration by Nox4

H. Kim, S. Ullevig, R. Asmis, 2013, PloS one.

Mitochondrion as a Target for Heart Failure Therapy- Role of Protein Lysine Acetylation.

R. Tian, C. F. Lee, 2015, Circulation journal : official journal of the Japanese Circulation Society.

Abstract 15979: Restoration of NADH/NAD Ratio Delays Pressure Overload-Induced Cardiac Dysfunction via Regulation of Mitochondrial Protein Acetylation and Permeability Transition

R. Tian, G. Karamanlidis, C. F. Lee, 2013 .

Late-life Rapamycin Treatment Enhances Cardiomyocyte Relaxation Kinetics and Reduces Myocardial Stiffness

P. Rabinovitch, Y. Chiao, K. Kobak, 2023, bioRxiv.