Songqing Lv

Long-term respiratory exposure to amorphous silica nanoparticles promoted systemic inflammation and progression of fibrosis in a susceptible mouse model.

Zhiwei Sun, Yan Li, Yanbo Li, 2022, Chemosphere.

Oxidative stress- and mitochondrial dysfunction-mediated cytotoxicity by silica nanoparticle in lung epithelial cells from metabolomic perspective.

Junchao Duan, Zhiwei Sun, Jie Zhang, 2021, Chemosphere.

PM2.5 triggered apoptosis in lung epithelial cells through the mitochondrial apoptotic way mediated by a ROS-DRP1-mitochondrial fission axis.

Zhiwei Sun, H. Duan, Yanbo Li, 2020, Journal of hazardous materials.

Biomarkers for the adverse effects on respiratory system health associated with atmospheric particulate matter exposure.

Yanbo Li, Yufan Liu, Caixia Guo, 2021, Journal of hazardous materials.

Amorphous silica nanoparticles caused lung injury through the induction of epithelial apoptosis via ROS/Ca2+/DRP1-mediated mitochondrial fission signaling

Zhiwei Sun, Yan Li, Yanbo Li, 2022, Nanotoxicology.

Disturbed mitochondrial quality control involved in hepatocytotoxicity induced by silica nanoparticles.

Zhiwei Sun, Yanbo Li, Alimire Abulikemu, 2020, Nanoscale.

Oxidative stress- and mitochondrial dysfunction-mediated cytotoxicity by silica nanoparticle in lung epithelial cells from metabolomic perspective

Junchao Duan, Zhiwei Sun, Jie Zhang, 2021, Chemosphere.