B. Ryu

发表

The Novel Neuroprotective Action of Sulfasalazine through Blockade of NMDA Receptors

Su-Youne Chang, Jun-Sub Choi, C. Joo, 2003, Journal of Pharmacology and Experimental Therapeutics.

Proapoptotic Effects of Tau Cleavage Product Generated by Caspase-3

D. Jo, I. Mook‐Jung, C. Chung, 2001, Neurobiology of Disease.

Role for PKC-ε in neuronal death induced by oxidative stress

I. Mook‐Jung, B. Lee, Yi-Sook Jung, 2004 .

Attenuation of cortical neuronal apoptosis by gangliosides.

E. Costa, D. Choi, I. Jou, 1999, The Journal of pharmacology and experimental therapeutics.

Attenuation of Cortical Neuronal Apoptosis by Gangliosides 1

E. Costa, D. Choi, I. Jou, 1999 .

Phosphatidylinositol 3-kinase-mediated regulation of neuronal apoptosis and necrosis by insulin and IGF-I.

I. Jou, H. Ko, B. Gwag, 1999, Journal of neurobiology.

A neuron-specific gene transfer by a recombinant defective Sindbis virus.

Y. Sung, H. Ko, B. Gwag, 1998, Brain research. Molecular brain research.

Blockade of ionotropic glutamate receptors produces neuronal apoptosis through the Bax‐cytochrome C‐caspase pathway: the causative role of Ca2+ deficiency

W. Yoon, B. Gwag, B. Ryu, 2003, Journal of neurochemistry.

Synergetic Activation of p38 Mitogen‐Activated Protein Kinase and Caspase‐3‐Like Proteases for Execution of Calyculin A‐Induced Apoptosis but Not N‐Methyl‐d‐Aspartate‐Induced Necrosis in Mouse Cortical Neurons

Y. Jung, Kong Sook Han, W. Yoon, 2000, Journal of neurochemistry.

Attenuation of Zn2+ Neurotoxicity by Aspirin: Role of N-Type Ca2+ Channel and the Carboxyl Acid Group

Su-Youne Chang, E. Y. Kim, C. Joo, 2001, Neurobiology of Disease.

Role for PKC-epsilon in neuronal death induced by oxidative stress.

I. Mook‐Jung, B. Lee, Yi-Sook Jung, 2004, Biochemical and Biophysical Research Communications - BBRC.

NT-4/5 Exacerbates Free Radical-Induced Neuronal Necrosis in Vitro and in Vivo

H. Ko, S. Sohn, Hyuk Jae Kwon, 2000, Neurobiology of Disease.

Phosphatidylinositol 3-kinase-mediated regulation of neuronal apoptosis and necrosis by insulin and IGF-I.

I. Jou, B. Gwag, J. Noh, 1999, Journal of neurobiology.