G. Prince

发表

Prevalence of Platelet Transfusion Reactions Before and After Implementation of Leukocyte‐Depleted Platelet Concentrates by Filtration

H. Lazarus, L. Goodnough, J. Riddell, 1993, Vox sanguinis.

Characterization of putative glycoinositol phospholipid anchor precursors in mammalian cells. Localization of phosphoethanolamine.

L. Ravi, L. Ravi, T. Rosenberry, 1992, The Journal of biological chemistry.

A candidate mammalian glycoinositol phospholipid precursor containing three phosphoethanolamines.

T. Rosenberry, M. E. Medof, S. Hirose, 1993, The Journal of biological chemistry.

A Simulation Analysis and Screening of Deleterious Non-Synonymous Single Nucleotide Polymorphisms (SNPs) in Human CDKN1A Gene

G. Prince, Trayee Dhar, 2017, bioRxiv.

The molecular basis for paroxysmal nocturnal hemoglobinuria

R. Yomtovian, G. Prince, M. Medof, 1993, Transfusion.

Structural properties of the glycoplasmanylinositol anchor phospholipid of the complement membrane attack complex inhibitor CD59

H. Okada, P. Lachmann, W. D. Ratnoff, 1992, Clinical and experimental immunology.

Peripheral blood harvest of unaffected CD34+ CD38- hematopoietic precursors in paroxysmal nocturnal hemoglobinuria.

H. Lazarus, L. Terstappen, R. Brodsky, 1995, Blood.

Restoration of normal interactions between immune complexes and systemic lupus erythematosus blood cells by normal serum.

J. Oger, J. Antel, G. Prince, 1982, Journal of clinical & laboratory immunology.

Effectiveness of RSVIG prophylaxis and therapy of respiratory syncytial virus in an immunosuppressed animal model

V. Hemming, D. Porter, M. Ottolini, 1999, Bone Marrow Transplantation.

A defect in glycosylphosphatidylinositol (GPI) transamidase activity in mutant K cells is responsible for their inability to display GPI surface proteins.

R. Chen, S. Udenfriend, G. Prince, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Release of soluble immune complexes from immune adherence receptors on human erythrocytes is mediated by C3b inactivator independently of Beta 1H and is accompanied by generation of C3c.

C. Mold, G. Prince, M. Medof, 1982, Proceedings of the National Academy of Sciences of the United States of America.

Impaired growth and elevated fas receptor expression in PIGA(+) stem cells in primary paroxysmal nocturnal hemoglobinuria.

H. Lazarus, R. Chen, D. Kaplan, 2000, The Journal of clinical investigation.

Prophylactic Administration of Respiratory Syncytial Virus Immune Globulin to High-Risk Infants and Young Children

C. Hall, D. Fulton, H. Meissner, 1993 .

Synthesis of mannosylglucosaminylinositol phospholipids in normal but not paroxysmal nocturnal hemoglobinuria cells.

M. Rosenfeld, L. Ravi, R. Silber, 1992, Proceedings of the National Academy of Sciences of the United States of America.

Differences in interaction between immune complexes and complement receptors in serum and cerebrospinal fluid.

J. Oger, J. Antel, B. Arnason, 1982, Clinical and Experimental Immunology.

Palivizumab is highly effective in suppressing respiratory syncytial virus in an immunosuppressed animal model

M. Ottolini, G. Prince, S. Curtis, 2002, Bone Marrow Transplantation.

Kinetics of interaction of immune complexes with complement receptors on human blood cells: modification of complexes during interaction with red cells.

J. Oger, G. Prince, M. Medof, 1982, Clinical and experimental immunology.

Requirement for human red blood cells in inactivation of C3b in immune complexes and enhancement of binding to spleen cells.

C. Mold, T. Lam, G. Prince, 1983, Journal of immunology.