E. Hammond

发表

Eruptive haemangioma associated with HIV therapy and mitochondrial pathology.

S. Mallal, Annalise M. Martin, D. Nolan, 2008, Pathology.

Rationale and design of the familial hypercholesterolemia foundation CAscade SCreening for Awareness and DEtection of Familial Hypercholesterolemia registry.

Emily C. O'Brien, J. Knowles, D. Rader, 2014, American heart journal.

Germline RBBP6 mutations in familial myeloproliferative neoplasms.

G. Superti-Furga, K. Bennett, A. Stukalov, 2016, Blood.

The role of the JAK2 GGCC haplotype and the TET2 gene in familial myeloproliferative neoplasms

M. Cazzola, C. Pascutto, R. Kralovics, 2011, Haematologica.

Concordance of assays designed for the quantification of JAK2V617F: a multicenter study

S. Ellard, J. Jelinek, N. Kröger, 2009, Haematologica.

External Quality Assessment of HLA-B*5701 Reporting: An International Multicentre Survey

S. Mallal, M. Gill, C. Mamotte, 2007, Antiviral therapy.

Role of international registries in enhancing the care of familial hypercholesterolaemia.

J. Knowles, G. Watts, Hanns Lochmüller, 2013, International journal of evidence-based healthcare.

Adipose tissue inflammation and altered adipokine and cytokine production in antiretroviral therapy-associated lipodystrophy

D. Nolan, E. Hammond, 2007, Current opinion in HIV and AIDS.

Longitudinal evaluation of cardiovascular disease-associated biomarkers in relation to abacavir therapy

S. Mallal, D. Nolan, E. McKinnon, 2008, AIDS.

NRTI therapy is associated with mitochondrial DNA depletion, increased mitochondrial biogenesis and subcutaneous fat wasting in HIV infected patients

M. John, S. Mallal, D. Nolan, 2001 .

Mitochondrial Proliferation, Dna Depletion and Adipocyte Differentiation in Subcutaneous Adipose Tissue of HIV-Positive Haart Recipients

S. Mallal, C. Mamotte, Annalise M. Martin, 2002, Antiviral therapy.

Human immunodeficiency virus treatment-induced adipose tissue pathology and lipoatrophy: prevalence and metabolic consequences.

D. Nolan, E. McKinnon, E. Hammond, 2010, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.

Differential gene expression indicates that ‘buffalo hump’ is a distinct adipose tissue disturbance in HIV-1-associated lipodystrophy

F. Villarroya, E. Martinez, M. Giralt, 2008, AIDS.

Assessing the contribution of ART, HIV and host factors to adipose tissue changes occurring in HIV-infected individuals: Risk profile for lipoatrophy

S. Mallal, D. Nolan, E. McKinnon, 2005 .

Mitochondrial DNA depletion and morphologic changes in adipocytes associated with nucleoside reverse transcriptase inhibitor therapy

S. Mallal, Annalise M. Martin, D. Nolan, 2003, AIDS.

Contribution of Nucleoside-Analogue Reverse Transcriptase Inhibitor Therapy to Lipoatrophy from the Population to the Cellular Level

I. James, S. Mallal, D. Nolan, 2002, Antiviral therapy.

IL28B, HLA-C, and KIR Variants Additively Predict Response to Therapy in Chronic Hepatitis C Virus Infection in a European Cohort: A Cross-Sectional Study

S. Mallal, G. Stewart, D. Booth, 2011, PLoS medicine.

IL 28 B , HLA-C , and KIR Variants Additively Predict Response to Therapy in Chronic Hepatitis C Virus Infection in a European Cohort : A Cross-Sectional Study

S. Mallal, G. Stewart, D. Booth, 2011 .

editor: Germline RBBP6 mutations in familial myeloproliferative neoplasms

G. Superti-Furga, K. Bennett, A. Stukalov, 2016 .

Germline RBBP6 Mutations In Myeloproliferative Neoplasms

G. Superti-Furga, K. Bennett, A. Stukalov, 2013 .

Cellular immune responses to HCV core increase and HCV RNA levels decrease during successful antiretroviral therapy

P. Klenerman, A. Telenti, H. Günthard, 2010, Gut.

Accumulation of mitochondrial DNA mutations in human immunodeficiency virus-infected patients treated with nucleoside-analogue reverse-transcriptase inhibitors.

F. Christiansen, S. Mallal, Annalise M. Martin, 2003, American journal of human genetics.

Assessment of precision and concordance of quantitative mitochondrial DNA assays: a collaborative international quality assurance study.

S. Mallal, J. Montaner, P. Reiss, 2003, Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology.

Subcutaneous fat tissue mitochondrial DNA depletion and adipose toxicity are strongly associated with nucleoside reverse transcriptase inhibitor (NRTI) therapy in HIV-infected patients

I. James, S. Mallal, D. Nolan, 2003 .

Quality assessment of human mitochondrial DNA quantification: MITONAUTS, an international multicentre survey.

F. Villarroya, Ò. Miró, P. Cinque, 2011, Mitochondrion.

Viral gastroenteritis – a danger to the patient, a danger to the staff

R. Appelboam, E. Hammond, 2004, Anaesthesia.

Reduction of mitochondrial DNA content and respiratory chain activity occurs in adipocytes within 6–12 months of commencing nucleoside reverse transcriptase inhibitor therapy

I. James, S. Mallal, D. Nolan, 2004, AIDS.

Taking Up Space: A Pedagogical History Of Flutists’ Perspectives On Breath And Support

E. Hammond, 2019 .

HLA-B*5701 typing: evaluation of an allele-specific polymerase chain reaction melting assay.

S. Mallal, C. Mamotte, D. Nolan, 2007, Tissue antigens.

Quantitative determination of JAK2 V617F by TaqMan: An absolute measure of averaged copies per cell that may be associated with the different types of myeloproliferative disorders.

Ian James, Richard Herrmann, Kathryn Shaw, 2007, The Journal of molecular diagnostics : JMD.

Key outcomes from stakeholder workshops at a symposium to inform the development of an Australian national plan for rare diseases

Caron Molster, H. Dawkins, C. Molster, 2012, Orphanet Journal of Rare Diseases.

The JAK2 V617F mutation is detectable in granulocyte populations at greater than two copies per cell among individuals with myeloproliferative disorders

R. Herrmann, K. Shaw, E. Hammond, 2007, Leukemia.

Rationale and design of the familial hypercholesterolemia foundation CAscade SCreening for Awareness and DEtection of Familial Hypercholesterolemia registry.

Emily C. O'Brien, J. Knowles, D. Rader, 2014, American heart journal.

“The JAK2 V617F mutation is detectable in granulocyte populations at greater than two copies per cell among individuals with myeloproliferative disorders”, by El Housni et al.

R. Herrmann, K. Shaw, E. Hammond, 2007, Leukemia.