Zhichao Zhou

发表

Erythrocytes From Patients With Type 2 Diabetes Induce Endothelial Dysfunction Via Arginase I.

C. Östenson, D. Andersson, U. Hedin, 2018, Journal of the American College of Cardiology.

Uridine Adenosine Tetraphosphate-Induced Coronary Relaxation Is Blunted in Swine With Pressure Overload: A Role for Vasoconstrictor Prostanoids

H. V. van Beusekom, D. Duncker, Caroline Cheng, 2018, Front. Pharmacol..

Involvement of NADPH oxidase in A2A adenosine receptor-mediated increase in coronary flow in isolated mouse hearts

C. Ledent, S. Tilley, Syed Jamal Mustafa, 2015, Purinergic Signalling.

vasomotor tone Nitroso-redox balance in control of coronary

L. Moens, D. Duncker, D. Merkus, 2015 .

Red Blood Cells in Type 2 Diabetes Impair Cardiac Post-Ischemic Recovery Through an Arginase-Dependent Modulation of Nitric Oxide Synthase and Reactive Oxygen Species

S. Catrina, K. Brismar, J. Lundberg, 2018, JACC. Basic to translational science.

Downregulation of Erythrocyte miR-210 Induces Endothelial Dysfunction in Type 2 Diabetes.

U. Hedin, S. Catrina, L. Maegdefessel, 2021, Diabetes.

miR‐499 released during myocardial infarction causes endothelial injury by targeting α7‐nAchR

F. Liu, Chenliang Deng, Xitong Dang, 2019, Journal of cellular and molecular medicine.

Adenosine and adenosine receptor-mediated action in coronary microcirculation

Yong Tang, B. Wernly, Ying Zhang, 2021, Basic Research in Cardiology.

Blunted coronary vasodilator response to uridine adenosine tetraphosphate in post-infarct remodeled myocardium is due to reduced P1 receptor activation.

J. Jankowski, V. Jankowski, D. Duncker, 2013, Pharmacological research.

Cytochrome P-450 2 C 9 exerts a vasoconstrictor influence on coronary resistance vessels in swine at rest and during exercise

D. Duncker, D. Merkus, V. Hemradj, 2012 .

Purinergic Dysfunction in Pulmonary Arterial Hypertension

C. Guignabert, D. Merkus, L. Tu, 2020, Journal of the American Heart Association.

Uridine adenosine tetraphosphate and purinergic signaling in cardiovascular system: An update

V. Jankowski, D. Duncker, D. Merkus, 2019, Pharmacological research.

Erythrocytes Induce Vascular Dysfunction in COVID-19

F. Farnebo, A. Färnert, J. Lundberg, 2021, JACC: Basic to Translational Science.

Sunitinib-Induced Systemic Vasoconstriction in Swine Is Endothelin Mediated and Does Not Involve Nitric Oxide or Oxidative Stress

S. Sleijfer, D. Duncker, A. Danser, 2012, Hypertension.

Erythrocytes from patients with ST-elevation myocardial infarction induce cardioprotection through the purinergic P2Y13 receptor and nitric oxide signaling

J. Jurga, J. Pernow, F. Böhm, 2022, Basic Research in Cardiology.

Uridine adenosine tetraphosphate is a novel vasodilator in the coronary microcirculation which acts through purinergic P1 but not P2 receptors.

D. Duncker, Caroline Cheng, D. Merkus, 2013, Pharmacological research.

Activation of adenosine A2A but not A2B receptors is involved in uridine adenosine tetraphosphate-induced porcine coronary smooth muscle relaxation

D. Duncker, D. Merkus, Changyan Sun, 2019, Journal of pharmacological sciences.

Altered purinergic signaling in uridine adenosine tetraphosphate-induced coronary relaxation in swine with metabolic derangement

D. Duncker, Caroline Cheng, D. Merkus, 2017, Purinergic Signalling.

Alteration of purinergic signaling in diabetes: Focus on vascular function.

R. Sprague, Xitong Dang, Syed Jamal Mustafa, 2020, Journal of molecular and cellular cardiology.

Phosphodiesterase 5 inhibition-induced coronary vasodilation is reduced after myocardial infarction.

D. Duncker, D. Merkus, B. Houweling, 2013, American journal of physiology. Heart and circulatory physiology.

Enrichment differentiation of human induced pluripotent stem cells into sinoatrial node-like cells by combined modulation of BMP, FGF, and RA signaling pathways

F. Liu, Xitong Dang, Linli Wang, 2020, Stem Cell Research & Therapy.

Sunitinib and its effect in the cardiovascular system.

J. Pernow, B. Wernly, Zhichao Zhou, 2021, Drug discovery today.

The red blood cell as a mediator of endothelial dysfunction in patients with familial hypercholesterolemia and dyslipidemia

Marcus Henricsson, J. Pernow, M. Levin, 2022, Journal of internal medicine.

Erythrocytes Induce Endothelial Injury in Type 2 Diabetes Through Alteration of Vascular Purinergic Signaling

J. Pernow, M. Alvarsson, Y. Tratsiakovich, 2020, Frontiers in Pharmacology.

Altered Purinergic Receptor Sensitivity in Type 2 Diabetes-Associated Endothelial Dysfunction and Up4A-Mediated Vascular Contraction

C. Östenson, J. Pernow, Y. Tratsiakovich, 2018, International journal of molecular sciences.

Purinergic interplay between erythrocytes and platelets in diabetes-associated vascular dysfunction

Zhichao Zhou, 2021, Purinergic Signalling.

Ticagrelor: a cardiometabolic drug targeting erythrocyte-mediated purinergic signaling?

D. Erlinge, J. Pernow, B. Wernly, 2020, American journal of physiology. Heart and circulatory physiology.

James Stray-Gundersen performance acclimatization with low-altitude training on ' ' Living high-training low ' ' : effect of moderate-altitude

Alain, Bruce D. Johnson, Matthew R. Ely, 2016 .

P2X7 Receptor–Mediated Inflammation in Cardiovascular Disease

Yong Tang, Xiaojing Liu, Junteng Zhou, 2021, Frontiers in Pharmacology.

Uridine adenosine tetraphosphate acts as a proangiogenic factor in vitro through purinergic P2Y receptors.

D. Duncker, Caroline Cheng, D. Merkus, 2016, American journal of physiology. Heart and circulatory physiology.

Red Blood Cell Peroxynitrite Causes Endothelial Dysfunction in Type 2 Diabetes Mellitus via Arginase

J. Pernow, B. Wernly, M. Alvarsson, 2020, Cells.

MicroRNA: A mediator of diet-induced cardiovascular protection.

J. Pernow, Hong Jin, Zhichao Zhou, 2021, Current opinion in pharmacology.

Red blood cell dysfunction: a new player in cardiovascular disease.

J. Pernow, Jiangning Yang, Zhichao Zhou, 2019, Cardiovascular research.

Anti-coagulation for COVID-19 treatment: both anti-thrombotic and anti-inflammatory?

U. Hoppe, M. Lichtenauer, A. Egle, 2020, Journal of Thrombosis and Thrombolysis.

Coronary microvascular dysfunction after long-term diabetes and hypercholesterolemia.

W. J. van der Giessen, D. Duncker, S. Koopmans, 2016, American journal of physiology. Heart and circulatory physiology.

Enhanced A2A adenosine receptor-mediated increase in coronary flow in type I diabetic mice.

Syed Jamal Mustafa, H. Labazi, B. Teng, 2016, Journal of molecular and cellular cardiology.

Therapeutic Potential of Sunitinib in Ameliorating Endothelial Dysfunction in Type 2 Diabetic Rats

C. Östenson, A. Danser, J. Pernow, 2021, Pharmacology.

Erythrocyte-Derived microRNAs: Emerging Players in Cardiovascular and Metabolic Disease

J. Pernow, Zhichao Zhou, Aida Collado, 2023, Arteriosclerosis, thrombosis, and vascular biology.

Divergent coronary flow responses to uridine adenosine tetraphosphate in atherosclerotic ApoE knockout mice

Syed Jamal Mustafa, H. Labazi, Xiao-rong Zeng, 2017, Purinergic Signalling.

Altered purinergic signaling in uridine adenosine tetraphosphate-induced coronary relaxation in swine with metabolic derangement

D. Duncker, Caroline Cheng, D. Merkus, 2017, Purinergic Signalling.

Mechanisms underlying uridine adenosine tetraphosphate-induced vascular contraction in mouse aorta: Role of thromboxane and purinergic receptors.

S. Tilley, Syed Jamal Mustafa, Changyan Sun, 2015, Vascular pharmacology.

A 15-Year Study on Up4A in Cardiovascular Disease

Zhichao Zhou, T. Matsumoto, 2020, Frontiers in Pharmacology.

Impaired Aortic Contractility to Uridine Adenosine Tetraphosphate in Angiotensin II-Induced Hypertensive Mice: Receptor Desensitization?

B. Teng, Changyan Sun, Zhichao Zhou, 2016, American journal of hypertension.

Involvement of NADPH oxidase in A2A adenosine receptor-mediated increase in coronary flow in isolated mouse hearts

C. Ledent, S. Tilley, Syed Jamal Mustafa, 2015, Purinergic Signalling.

Cytochrome P-450 2C9 exerts a vasoconstrictor influence on coronary resistance vessels in swine at rest and during exercise.

D. Duncker, D. Merkus, V. Hemradj, 2012, American journal of physiology. Heart and circulatory physiology.

Phosphodiesterase-5 activity exerts a coronary vasoconstrictor influence in awake swine that is mediated in part via an increase in endothelin production.

D. Duncker, S. Bender, D. Merkus, 2014, American journal of physiology. Heart and circulatory physiology.

Purinergic activation in response to hemodynamic force directs heart valve development

Xin Cao, Zhichao Zhou, 2022, Purinergic Signalling.

Pulmonary vasoconstrictor influence of endothelin in exercising swine depends critically on phosphodiesterase 5 activity.

D. Duncker, S. Bender, Daphne P M de Wijs-Meijler, 2014, American journal of physiology. Lung cellular and molecular physiology.

The Effect of Glycemic Control on Endothelial and Cardiac Dysfunction Induced by Red Blood Cells in Type 2 Diabetes

J. Pernow, M. Alvarsson, Jiangning Yang, 2019, Front. Pharmacol..

Erythrocytes and cardiovascular complications

J. Pernow, Jiangning Yang, Zhichao Zhou, 2018, Aging.

Inhibition of Long Noncoding RNA Gm41724 Alleviates Pressure Overload-induced Cardiac Fibrosis by Regulating Lamin-Associated Polypeptide 2α.

Wenchao Wu, Junteng Zhou, Qihang Kong, 2023, Pharmacological research.

Extracellular vesicles and their non‐coding RNA cargos: Emerging players in cardiovascular disease

L. Gan, J. Pernow, Zhichao Zhou, 2022, The Journal of physiology.

Phosphodiesterase 5 inhibition-induced coronary vasodilation is reduced after myocardial infarction.

D. Duncker, D. Merkus, B. Houweling, 2013, American journal of physiology. Heart and circulatory physiology.

Mechanisms underlying uridine adenosine tetraphosphate-induced vascular contraction in mouse aorta: Role of thromboxane and purinergic receptors.

S. Tilley, Syed Jamal Mustafa, Changyan Sun, 2015, Vascular pharmacology.

Hypoxic erythrocytes mediate cardioprotection through activation of soluble guanylate cyclase and release of cyclic GMP

S. Catrina, E. Weitzberg, J. Lundberg, 2023, The Journal of clinical investigation.