L. Mullan

Stimulation of intracellular proteolytic degradation as a means of reducing ER stress in a model of skeletal dysplasia

L. Mullan, 2015 .

Armet/Manf and Creld2 are components of a specialized ER stress response provoked by inappropriate formation of disulphide bonds: implications for genetic skeletal diseases

R. Boot-Handford, M. Briggs, Sarah Edwards, 2013, Human molecular genetics.

XBP1-Independent UPR Pathways Suppress C/EBP-β Mediated Chondrocyte Differentiation in ER-Stress Related Skeletal Disease

L. Glimcher, R. Boot-Handford, J. Bateman, 2015, PLoS genetics.

Cartilage endoplasmic reticulum stress may influence the onset but not the progression of experimental osteoarthritis

R. Boot-Handford, Ping Wang, J. Soul, 2019, Arthritis Research & Therapy.

Increased intracellular proteolysis reduces disease severity in an ER stress–associated dwarfism

R. Goodacre, R. Boot-Handford, J. Bateman, 2017, The Journal of clinical investigation.