P. M. Bryan

Down-Regulation Does Not Mediate Natriuretic Peptide-Dependent Desensitization of Natriuretic Peptide Receptor (NPR)-A or NPR-B: Guanylyl Cyclase-Linked Natriuretic Peptide Receptors Do Not Internalize

L. R. Potter, Danhua Fan, Laura K. Antos, 2005, Molecular Pharmacology.

Differential regulation of membrane guanylyl cyclases in congestive heart failure: natriuretic peptide receptor (NPR)-B, Not NPR-A, is the predominant natriuretic peptide receptor in the failing heart.

Xin Xu, Yingjie Chen, D. Dickey, 2007, Endocrinology.

Renal hyporesponsiveness to atrial natriuretic peptide in congestive heart failure results from reduced atrial natriuretic peptide receptor concentrations.

Xin Xu, Yingjie Chen, D. Dickey, 2007, American journal of physiology. Renal physiology.

The atrial natriuretic peptide receptor (NPR-A/GC-A) is dephosphorylated by distinct microcystin-sensitive and magnesium-dependent protein phosphatases

P. M. Bryan, L. Potter, 2003 .

A sensitive method for determining the phosphorylation status of natriuretic peptide receptors: cGK‐I alpha does not regulate NPR‐A

F. Hofmann, R. Feil, S. Lohmann, 2006, Biochemistry.

The Atrial Natriuretic Peptide Receptor (NPR-A/GC-A) Is Dephosphorylated by Distinct Microcystin-sensitive and Magnesium-dependent Protein Phosphatases*

P. M. Bryan, L. Potter, 2002, The Journal of Biological Chemistry.

Artificial Lithium Toxicity

A. Dzierba, Subani Chandra, K. Connolly, 2015, Journal of pharmacy practice.