D. Kimono

发表

Increased butyrate priming in the gut stalls microbiome associated‐gastrointestinal inflammation and hepatic metabolic reprogramming in a mouse model of Gulf War Illness

M. Nagarkatti, P. Nagarkatti, S. Chatterjee, 2018, Toxicology and applied pharmacology.

Microcystin exposure worsens nonalcoholic fatty liver disease associated ectopic glomerular toxicity via NOX-2-MIR21 axis.

M. Nagarkatti, P. Nagarkatti, D. Porter, 2019, Environmental toxicology and pharmacology.

High circulatory leptin mediated NOX-2-peroxynitrite-miR21 axis activate mesangial cells and promotes renal inflammatory pathology in nonalcoholic fatty liver disease

M. Nagarkatti, P. Nagarkatti, S. Raychoudhury, 2018, Redox Biology.

Early Microcystin-LR exposure-linked inflammasome activation in mice causes development of fatty liver disease and insulin resistance.

M. Nagarkatti, P. Nagarkatti, D. Porter, 2020, Environmental toxicology and pharmacology.

Exogenous PP2A inhibitor exacerbates progression of nonalcoholic fatty liver disease via NOX2 dependent activation of miR21.

M. Nagarkatti, P. Nagarkatti, D. Porter, 2019, American journal of physiology. Gastrointestinal and liver physiology.

Sparstolonin B (SsnB) attenuates liver fibrosis via a parallel conjugate pathway involving P53‐P21 axis, TGF‐beta signaling and focal adhesion that is TLR4 dependent

M. Nagarkatti, P. Nagarkatti, A. Diehl, 2018, European journal of pharmacology.

TLR Antagonism by Sparstolonin B Alters Microbial Signature and Modulates Gastrointestinal and Neuronal Inflammation in Gulf War Illness Preclinical Model

M. Nagarkatti, P. Nagarkatti, N. Klimas, 2020, Brain sciences.

Environmental microcystin targets the microbiome and increases the risk of intestinal inflammatory pathology via NOX2 in underlying murine model of Nonalcoholic Fatty Liver Disease

M. Nagarkatti, P. Nagarkatti, D. Porter, 2019, Scientific Reports.

Higher intestinal and circulatory lactate associated NOX2 activation leads to an ectopic fibrotic pathology following microcystin co-exposure in murine fatty liver disease.

M. Nagarkatti, P. Nagarkatti, D. Porter, 2020, Comparative biochemistry and physiology. Toxicology & pharmacology : CBP.

Host Akkermansia muciniphila Abundance Correlates With Gulf War Illness Symptom Persistence via NLRP3-Mediated Neuroinflammation and Decreased Brain-Derived Neurotrophic Factor

N. Klimas, S. Chatterjee, R. Seth, 2020, Neuroscience insights.

Effects of boiling solution (trona) concentration and time on the proximate composition and physico- chemical propterties of flour from three-leaved yam (Dioscorea dumetorum pax) tubers

G. W. Byarugaba-Bazirake, W. Byarugaba, D. Kimono, 2011 .

Gut DNA Virome Diversity and Its Association with Host Bacteria Regulate Inflammatory Phenotype and Neuronal Immunotoxicity in Experimental Gulf War Illness

N. Klimas, R. Maqsood, E. Lim, 2019, Viruses.

Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis

M. Nagarkatti, P. Nagarkatti, S. Chatterjee, 2020, Journal of Neuroinflammation.

Environmental microcystin targets the microbiome and increases the risk of intestinal inflammatory pathology via NOX2 in underlying murine model of Nonalcoholic Fatty Liver Disease

M. Nagarkatti, P. Nagarkatti, D. Porter, 2019, Scientific Reports.

Dysbiosis-Associated Enteric Glial Cell Immune-Activation and Redox Imbalance Modulate Tight Junction Protein Expression in Gulf War Illness Pathology

M. Nagarkatti, P. Nagarkatti, N. Klimas, 2019, Front. Physiol..

Gastrointestinal problems, mechanisms and possible therapeutic directions in Gulf war illness: a mini review

D. Kimono, 2021, Military Medical Research.

Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis

M. Nagarkatti, P. Nagarkatti, S. Chatterjee, 2020, Journal of Neuroinflammation.

Exogenous PP2A inhibitor exacerbates the progression of nonalcoholic fatty liver disease via NOX2-dependent activation of miR21

M. Nagarkatti, P. Nagarkatti, D. Porter, 2019, American Journal of Physiology-Gastrointestinal and Liver Physiology.

A Population of Tumor-Infiltrating CD4+ T-cells Co-expressing CD38 and CD39 is Associated with Checkpoint Inhibitor Resistance.

David M. Woods, J. Weber, A. Puranik, 2023, Clinical cancer research : an official journal of the American Association for Cancer Research.