E. Frey

发表

DLK Activation Synergizes with Mitochondrial Dysfunction to Downregulate Axon Survival Factors and Promote SARM1-Dependent Axon Degeneration

J. Milbrandt, A. Diantonio, Daniel W. Summers, 2019, Molecular Neurobiology.

Cytoskeletal disruption activates the DLK/JNK pathway, which promotes axonal regeneration and mimics a preconditioning injury

A. Diantonio, V. Valakh, E. Babetto, 2015, Neurobiology of Disease.

Structure and activity of the acid-sensing ion channels.

C. Askwith, T. Sherwood, E. Frey, 2012, American journal of physiology. Cell physiology.

DLK Activation Synergizes with Mitochondrial Dysfunction to Downregulate Axon Survival Factors and Promote SARM1-Dependent Axon Degeneration

J. Milbrandt, A. Diantonio, Daniel W. Summers, 2019, Molecular Neurobiology.

Conformational Changes in the Lower Palm Domain of ASIC1a Contribute to Desensitization and RFamide Modulation

R. Pavlovicz, Chenglong Li, C. Askwith, 2013, PloS one.

An in vitro assay to study induction of the regenerative state in sensory neurons

J. Milbrandt, A. Diantonio, V. Valakh, 2015, Experimental Neurology.

HSP90 is a chaperone for DLK and is required for axon injury signaling

J. Milbrandt, A. Diantonio, A. Russo, 2018, Proceedings of the National Academy of Sciences.

An evolutionarily conserved mechanism for cAMP elicited axonal regeneration involves direct activation of the dual leucine zipper kinase DLK

Catherine A. Collins, L. Holzman, A. Diantonio, 2016, eLife.

TRPV1 Agonist, Capsaicin, Induces Axon Outgrowth after Injury via Ca2+/PKA Signaling

J. Milbrandt, A. Diantonio, E. Frey, 2018, eNeuro.