D. Decraene

A low UVB dose, with the potential to trigger a protective p53-dependent gene program, increases the resilience of keratinocytes against future UVB insults.

L. Declercq, D. Maes, M. Garmyn, 2005, The Journal of investigative dermatology.

A synthetic superoxide dismutase/catalase mimetic (EUK-134) inhibits membrane-damage-induced activation of mitogen-activated protein kinase pathways and reduces p53 accumulation in ultraviolet B-exposed primary human keratinocytes.

L. Declercq, D. Maes, M. Garmyn, 2004, The Journal of investigative dermatology.

Acute response of human skin to solar radiation: regulation and function of the p53 protein.

P. Agostinis, M. Garmyn, P. de Haes, 2001, Journal of photochemistry and photobiology. B, Biology.

p53 protein and the response of human skin to UV

P. Agostinis, M. Garmyn, D. Decraene, 2001 .

AKT delays the early-activated apoptotic pathway in UVB-irradiated keratinocytes via BAD translocation.

P. Agostinis, M. Garmyn, A. Van Laethem, 2007, The Journal of investigative dermatology.

AKT status controls susceptibility of malignant keratinocytes to the early-activated and UVB-induced apoptotic pathway.

P. Agostinis, H. Degreef, M. Garmyn, 2004, The Journal of investigative dermatology.

Insulin-like Growth Factor-1-mediated AKT Activation Postpones the Onset of Ultraviolet B-induced Apoptosis, Providing More Time for Cyclobutane Thymine Dimer Removal in Primary Human Keratinocytes*

P. Agostinis, H. Degreef, M. Garmyn, 2002, The Journal of Biological Chemistry.

Conventional and Portable X-ray Fluorescence Determination of Ruthenium on Catalytic Surfaces

W. R. Bramstedt, D. Harrington, D. Decraene, 1976 .