L. Simmons

Mechanism of impaired responses of cerebral arterioles during diabetes mellitus.

W. Mayhan, L. Simmons, G. Sharpe, 1991, The American journal of physiology.

Evaluation of Antibiotic Efficacy Using Electron Microscopy: Morphological Effects of Guanylureido Cephalosporin, BL-P1654, and Carbenicillin on Escherichia coli

D. K. Herron, D. Preston, L. Simmons, 1976, Antimicrobial Agents and Chemotherapy.

Secondary structure of amyloid beta peptide correlates with neurotoxic activity in vitro.

D. Brems, R. Rydel, I. Lieberburg, 1994, Molecular pharmacology.

Substance P modulates single-channel properties of neuronal nicotinic acetylcholine receptors

S. Schuetze, L. Role, L. Simmons, 1990, Neuron.

β-amyloid peptide in vitro toxicity: Lot-to-lot variability

P. May, G. Becker, B. Gitter, 1992, Neurobiology of Aging.

Activation of a caspase 3-related cysteine protease is required for glutamate-mediated apoptosis of cultured cerebellar granule neurons.

S. Paul, R. Dodel, K. Bales, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Three mutations in v-Rel render it resistant to cleavage by cell-death protease caspase-3.

J. Scarpati, T. Gilmore, M. Barkett, 2001, Biochimica et biophysica acta.

Serotonin enhances the excitatory acetylcholine response in the RB cell cluster of Aplysia californica

J. Koester, L. Simmons, 1986, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Neuronal membrane conductance activated by amyloid β peptide: importance of peptide conformation

D. Czilli, L. Simmons, Wei Ying Li, 1995, Brain Research.

Suppression of an amyloid β-peptidemediated calcium channel response by a secreted β-amyloid precursor protein

J. Hale, D. Mcclure, Wanda Li, 1999, Neuroscience.