In Hee Lee

A novel non-agonist PPARγ ligand UHC1 blocks PPARγ phosphorylation by CDK5 and improves insulin sensitivity. Running title: Novel anti-diabetic non-agonist PPARγ ligand

Yong Ryoul Yang, J. Choi, P. Suh, 2014 .

Endogenous glucocorticoids and impaired insulin signaling are both required to stimulate muscle wasting under pathophysiological conditions in mice.

W. Mitch, Zhaoyong Hu, Jie Du, 2009, The Journal of clinical investigation.

PTEN Expression Contributes to the Regulation of Muscle Protein Degradation in Diabetes

W. Mitch, Zhaoyong Hu, Jie Du, 2007, Diabetes.

PTEN Inhibition Improves Muscle Regeneration in Mice Fed a High-Fat Diet

W. Mitch, Zhaoyong Hu, Jie Du, 2010, Diabetes.

A Novel Non-agonist Peroxisome Proliferator-activated Receptor γ (PPARγ) Ligand UHC1 Blocks PPARγ Phosphorylation by Cyclin-dependent Kinase 5 (CDK5) and Improves Insulin Sensitivity*

Yong Ryoul Yang, J. Choi, P. Suh, 2014, The Journal of Biological Chemistry.

PPARγ Antagonist Gleevec Improves Insulin Sensitivity and Promotes the Browning of White Adipose Tissue

P. Griffin, Yong Ryoul Yang, J. Choi, 2016, Diabetes.