N. M. Saw

The Roles of the Voa Subunit of the Vacuolar H+-ATPase in Dense-core Vesicle Acidification, Transmitter Uptake and Storage

N. M. Saw, 2011 .

Calcium-dependent Activator Protein for Secretion 1 (CAPS1) Binds to Syntaxin-1 in a Distinct Mode from Munc13-1*

Seungmee Park, S. Sugita, Lijun Li, 2013, The Journal of Biological Chemistry.

Control of Long-Term Synaptic Potentiation and Learning by Alternative Splicing of the NMDA Receptor Subunit GluN1.

Arun K. Ramani, S. Scherer, M. Brudno, 2019, Cell reports.

LIMK1 Regulates Long-Term Memory and Synaptic Plasticity via the Transcriptional Factor CREB

O. Snead, M. Cortez, Zhengping Jia, 2015, Molecular and Cellular Biology.

Vacuolar H+-ATPase subunits Voa1 and Voa2 cooperatively regulate secretory vesicle acidification, transmitter uptake, and storage

Y. Wada, G. Sun-Wada, S. Sugita, 2011, Molecular biology of the cell.

Munc18-1 domain-1 controls vesicle docking and secretion by interacting with syntaxin-1 and chaperoning it to the plasma membrane

Nancy T. Malintan, B. Collins, F. Meunier, 2011, Molecular biology of the cell.

The role of HDL and its modulators in the development of diabetes

D. Ng, N. M. Saw, 2012, Current opinion in lipidology.

LIMK1 Regulates Long-Term Memory and Synaptic Plasticity via the Transcriptional Factor CREB

Zhengping Jia, S. Asrar, Wei Xie, 2015, Molecular and Cellular Biology.