M. Chang

Histone H3 lysine 79 methyltransferase Dot1 is required for immortalization by MLL oncogenes.

N. Zeleznik-Le, Wenzheng Zhang, C. Chou, 2010, Cancer research.

Feedback regulation mediated by Bcl-2 and DARPP-32 regulates inositol 1,4,5-trisphosphate receptor phosphorylation and promotes cell survival

M. Berridge, J. Parys, F. Zhong, 2014, Proceedings of the National Academy of Sciences.

Molecular and Cellular Pathobiology Histone H 3 Lysine 79 Methyltransferase Dot 1 Is Required for Immortalization by MLL Oncogenes

N. Zeleznik-Le, Wenzheng Zhang, C. Chou, 2010 .

Importance of a specific amino acid pairing for murine MLL leukemias driven by MLLT1/3 or AFF1/4.

N. Zeleznik-Le, Shubin Zhang, J. Bushweller, 2014, Leukemia research.

Characterization of specific roles for AF4 and Dot1 in transformation mediated by MLL-AF9 leukemic oncoprotein

M. Chang, Ming-jin Chang, 2011 .

Acidosis Sensing Receptor GPR65 Correlates with Anti-Apoptotic Bcl-2 Family Member Expression in CLL Cells: Potential Implications for the CLL Microenvironment

A. Sattar, A. Almasan, B. Hill, 2014, Journal of leukemia.

A synthetic peptide targeting the BH4 domain of Bcl-2 induces apoptosis in multiple myeloma and follicular lymphoma cells alone or in combination with agents targeting the BH3-binding pocket of Bcl-2

J. Pink, S. Matsuyama, E. Greenberg, 2015, Oncotarget.