R. Terry

发表

How much dystrophin is enough: the physiological consequences of different levels of dystrophin in the mdx mouse.

S. Hammond, M. Wood, S. El Andaloussi, 2015, Human molecular genetics.

Poloxomer 188 Has a Deleterious Effect on Dystrophic Skeletal Muscle Function

D. Wells, R. Terry, H. Kaneb, 2014, PloS one.

Preventing phosphorylation of dystroglycan ameliorates the dystrophic phenotype in mdx mouse.

S. Winder, D. Wells, C. Moore, 2012, Human molecular genetics.

Histopathological Evaluation of Skeletal Muscle with Specific Reference to Mouse Models of Muscular Dystrophy

D. Wells, R. Terry, 2016, Current protocols in mouse biology.

Computed tomographic arthrography, gross anatomy and histology demonstrate a communication between synovial invaginations in the proximal aspect of the third interosseous muscle and the carpometacarpal joint in horses

R. Terry, Roger Smith, A. Foote, 2022, Frontiers in Veterinary Science.

Transgenic Mouse Proteomics Identifies New 14-3-3-associated Proteins Involved in Cytoskeletal Rearrangements and Cell Signaling*S

Miro Brajenovic, G. Drewes, T. Bouwmeester, 2006, Molecular & Cellular Proteomics.

Genome sequencing reveals a splice donor site mutation in the SNX14 gene associated with a novel cerebellar cortical degeneration in the Hungarian Vizsla dog breed

C. Mellersh, M. Boursnell, O. Forman, 2016, BMC Genetics.

The transgenic expression of LARGE exacerbates the muscle phenotype of dystroglycanopathy mice.

Susan C. Brown, F. Muntoni, Jihee Kim, 2014, Human molecular genetics.

Genome sequencing reveals a splice donor site mutation in the SNX14 gene associated with a novel cerebellar cortical degeneration in the Hungarian Vizsla dog breed

C. Mellersh, M. Boursnell, O. Forman, 2016, BMC Genetics.