Hideto Mishina

发表

Predictive value of serial assessment of endothelial function in chronic heart failure.

Takamitsu Nakamura, D. Fujioka, Yosuke Watanabe, 2012, International journal of cardiology.

Comparative study of bezafibrate and pravastatin in patients with coronary artery disease and high levels of remnant lipoprotein.

Takamitsu Nakamura, D. Fujioka, Yosuke Watanabe, 2010, Circulation journal : official journal of the Japanese Circulation Society.

Endothelial vasomotor dysfunction in the brachial artery predicts the short-term development of early stage renal dysfunction in patients with coronary artery disease.

J. Obata, Takamitsu Nakamura, D. Fujioka, 2011, International journal of cardiology.

Mice lacking the glutamate-cysteine ligase modifier subunit are susceptible to myocardial ischaemia-reperfusion injury.

J. Obata, Takamitsu Nakamura, D. Fujioka, 2010, Cardiovascular Research.

Chronic depletion of glutathione exacerbates ventricular remodelling and dysfunction in the pressure-overloaded heart.

J. Obata, Takamitsu Nakamura, D. Fujioka, 2013, Cardiovascular research.

Disruption of group IVA cytosolic phospholipase A(2) attenuates myocardial ischemia-reperfusion injury partly through inhibition of TNF-α-mediated pathway.

Takao Shimizu, J. Obata, Takamitsu Nakamura, 2012, American journal of physiology. Heart and circulatory physiology.

Group X secretory PLA2 in neutrophils plays a pathogenic role in abdominal aortic aneurysms in mice.

J. Obata, Takamitsu Nakamura, D. Fujioka, 2012, American journal of physiology. Heart and circulatory physiology.

secretory PLA 2 in neutrophils plays a pathogenic role in abdominal aortic aneurysms in mice

J. Obata, Takamitsu Nakamura, D. Fujioka, 2011 .

This information is current as Inflammation Exacerbates Ovalbumin-Induced Lung Receptor 2 Deficiency of Phospholipase

M. Murakami, J. Obata, Takamitsu Nakamura, 2013 .

Lack of Phospholipase a 2 Receptor Increases Susceptibility to Cardiac Rupture after Myocardial Infarction Running Title: Pla 2 Receptor and Myocardial Infarction

M. Murakami, J. Obata, Takamitsu Nakamura, 2022 .

Lack of Phospholipase A 2 Receptor Increases Susceptibility to Cardiac Rupture After Myocardial Infarction

M. Murakami, J. Obata, Takamitsu Nakamura, 2014 .

Group V secretory phospholipase A2 plays a pathogenic role in myocardial ischaemia-reperfusion injury.

J. Obata, Takamitsu Nakamura, D. Fujioka, 2011, Cardiovascular research.

Predictive value of remnant lipoprotein for cardiovascular events in patients with coronary artery disease after achievement of LDL-cholesterol goals.

J. Obata, Takamitsu Nakamura, Yosuke Watanabe, 2011, Atherosclerosis.

Contrast-enhanced ultrasound imaging of carotid plaque neovascularization is useful for identifying high-risk patients with coronary artery disease.

J. Obata, Takamitsu Nakamura, Yosuke Watanabe, 2013, Circulation journal : official journal of the Japanese Circulation Society.

Inhibition of Secretory Phospholipase A2 Activity Attenuates Acute Cardiogenic Pulmonary Edema Induced by Isoproterenol Infusion in Mice After Myocardial Infarction

J. Obata, Takamitsu Nakamura, Yosuke Watanabe, 2010, Journal of cardiovascular pharmacology.

Phospholipase A2 expression in coronary thrombus is increased in patients with recurrent cardiac events after acute myocardial infarction.

J. Obata, Takamitsu Nakamura, Yosuke Watanabe, 2013, International journal of cardiology.

Lack of Phospholipase A2 Receptor Increases Susceptibility to Cardiac Rupture After Myocardial Infarction

M. Murakami, J. Obata, Takamitsu Nakamura, 2014, Circulation research.

This information is current as Inflammation Exacerbates Ovalbumin-Induced Lung Receptor 2 Deficiency of Phospholipase

M. Murakami, J. Obata, Takamitsu Nakamura, 2013 .