À. Marzal-Casacuberta

Functional Lecithin:Cholesterol Acyltransferase Deficiency and High Density Lipoprotein Deficiency in Transgenic Mice Overexpressing Human Apolipoprotein A-II (*)

F. González-Sastre, L. Chan, F. Blanco-Vaca, 1996, The Journal of Biological Chemistry.

Human apolipoprotein A-II is a pro-atherogenic molecule when it is expressed in transgenic mice at a level similar to that in humans: evidence of a potentially relevant species-specific interaction with diet.

J. Ordóñez‐Llanos, F. González-Sastre, L. Chan, 1998, Journal of lipid research.

ApoA-II expression in CETP transgenic mice increases VLDL production and impairs VLDL clearance.

J. Ordóñez‐Llanos, F. González-Sastre, F. Blanco-Vaca, 2001, Journal of lipid research.

Increased production of very-low-density lipoproteins in transgenic mice overexpressing human apolipoprotein A-II and fed with a high-fat diet.

J. Ordóñez‐Llanos, F. González-Sastre, F. Blanco-Vaca, 2000, Biochimica et biophysica acta.

Molecular basis of fish-eye disease in a patient from Spain. Characterization of a novel mutation in the LCAT gene and lipid analysis of the cornea.

S. Qu, H. Pownall, I. Hurtado, 1997, Arteriosclerosis, thrombosis, and vascular biology.

Expression of human apolipoprotein A-II in apolipoprotein E-deficient mice induces features of familial combined hyperlipidemia.

J. Ordóñez‐Llanos, F. González-Sastre, F. Blanco-Vaca, 2000, Journal of lipid research.

Free cholesterol deposition in the cornea of human apolipoprotein A-II transgenic mice with functional lecithin: cholesterol acyltransferase deficiency.

F. González-Sastre, F. Blanco-Vaca, R. Casaroli-Marano, 1999, Metabolism: clinical and experimental.