S. Harenkamp

Trafficking-deficient long QT syndrome mutation KCNQ1-T587M confers severe clinical phenotype by impairment of KCNH2 membrane localization: evidence for clinically significant IKr-IKs alpha-subunit interaction.

S. Hohnloser, S. Nattel, R. Brandes, 2009, Heart rhythm.

Cellular properties of C-terminal KCNH2 long QT syndrome mutations: description and divergence from clinical phenotypes.

S. Hohnloser, S. Nattel, R. Brandes, 2008, Heart rhythm.

Preanalytic depletion of medicinal anti-CD38 antibody from patient plasma for immunohematology testing

E. Seifried, C. Geisen, H. Bonig, 2021, Blood.

Adaptive Immunity and Pathogenesis of Diabetes: Insights Provided by the α4–Integrin Deficient NOD Mouse

A. Stenzinger, T. Papayannopoulou, N. Müller, 2020, Cells.

The NADPH Oxidase Nox4 mediates tumour angiogenesis

M. Mittelbronn, K. Schröder, J. Epah, 2016, Acta physiologica.

Hematopoietic-Extrinsic Cues Dictate Circadian Redistribution of Mature and Immature Hematopoietic Cells in Blood and Spleen

Erik Maronde, M. Pfeffer, E. Wiercinska, 2019, Cells.

The Cytosolic NADPH Oxidase Subunit NoxO1 Promotes an Endothelial Stalk Cell Phenotype

R. Brandes, N. Weissmann, M. Mittelbronn, 2016, Arteriosclerosis, thrombosis, and vascular biology.

Knock out of the NADPH oxidase Nox4 has no impact on life span in mice

N. Weissmann, K. Schröder, M. Seimetz, 2016, Redox biology.