B. Ceacareanu

发表

Gab 1 , SHP 2 , and Protein Kinase A Are Crucial for the Activation of the Endothelial NO Synthase by Fluid Shear Stress

R. Busse, M. Dixit, B. Fisslthaler, 2005 .

role of PTP-PEST and p130cas muscle cell motility by decreasing Rac1 activity: essential Nitric oxide attenuates IGF-I-induced aortic smooth

C. Waters, A. Hassid, D. Zhuang, 2013 .

Nitric oxide attenuates IGF-I-induced aortic smooth muscle cell motility by decreasing Rac1 activity: essential role of PTP-PEST and p130cas.

C. Waters, A. Hassid, R. Ray, 2006, American journal of physiology. Cell physiology.

Nitric oxide attenuates insulin- or IGF-I-stimulated aortic smooth muscle cell motility by decreasing H2O2 levels: essential role of cGMP.

C. Waters, G. Rao, M. Dixit, 2004, American journal of physiology. Heart and circulatory physiology.

Chronic insulin treatment amplifies PDGF-induced motility in differentiated aortic smooth muscle cells by suppressing the expression and function of PTP1B.

M. Dixit, A. Hassid, D. Zhuang, 2008, American journal of physiology. Heart and circulatory physiology.

Nitric Oxide-Induced Motility in Aortic Smooth Muscle Cells: Role of Protein Tyrosine Phosphatase SHP-2 and GTP-Binding Protein Rho

N. Sreejayan, M. Dixit, A. Hassid, 2002, Circulation research.

Smooth Muscle Cells : Dependence on Gab1 and Gab1-shp2 Association Treatment with Insulin Uncovers the Motogenic Capacity of Nitric Oxide in Aortic

M. Dixit, A. Hassid, D. Zhuang, 2022 .

Regulation of cell motility by tyrosine phosphorylated villin.

C. Waters, A. Hassid, Alok Tomar, 2004, Molecular biology of the cell.

Gab1, SHP2, and Protein Kinase A Are Crucial for the Activation of the Endothelial NO Synthase by Fluid Shear Stress

R. Busse, M. Dixit, B. Fisslthaler, 2005, Circulation research.

Essential role of protein kinase G and decreased cytoplasmic Ca2+ levels in NO-induced inhibition of rat aortic smooth muscle cell motility.

A. Hassid, D. Zhuang, A. Ceacareanu, 2005, American journal of physiology. Heart and circulatory physiology.

Counter-Regulatory Function of Protein Tyrosine Phosphatase 1B in Platelet-Derived Growth Factor– or Fibroblast Growth Factor–Induced Motility and Proliferation of Cultured Smooth Muscle Cells and in Neointima Formation

Chunxiang Zhang, A. Hassid, D. Zhuang, 2005, Arteriosclerosis, thrombosis, and vascular biology.

RhoA and Rac1 are both required for efficient wound closure of airway epithelial cells.

C. Waters, A. Hassid, L. Desai, 2004, American journal of physiology. Lung cellular and molecular physiology.