T. Desta

发表

Diabetes enhances mRNA levels of proapoptotic genes and caspase activity, which contribute to impaired healing.

D. Graves, Rongkun Liu, Y. Behl, 2006, Diabetes.

Diabetes alters the response to bacteria by enhancing fibroblast apoptosis.

D. Graves, Hongbin He, Rongkun Liu, 2004, Endocrinology.

Altered Fibroblast Proliferation and Apoptosis in Diabetic Gingival Wounds

D. Graves, T. Chino, T. Desta, 2010, Journal of dental research.

Tumor Necrosis Factor- (cid:1) Mediates Diabetes-Enhanced Apoptosis of Matrix-Producing Cells and Impairs Diabetic Healing

D. Graves, Rongkun Liu, Y. Behl, 2022 .

Diabetes prolongs the inflammatory response to a bacterial stimulus through cytokine dysregulation.

D. Graves, G. Naguib, H. Al-Mashat, 2004, The Journal of investigative dermatology.

Immunization Enhances Inflammation and Tissue Destruction in Response to Porphyromonas gingivalis

D. Graves, S. Amar, M. Siqueira, 2006, Infection and Immunity.

Impaired wound healing in mouse models of diabetes is mediated by TNF-α dysregulation and associated with enhanced activation of forkhead box O1 (FOXO1)

D. Graves, M. Siqueira, M. Alikhani, 2010, Diabetologia.

Diabetes-enhanced tumor necrosis factor-alpha production promotes apoptosis and the loss of retinal microvascular cells in type 1 and type 2 models of diabetic retinopathy.

D. Graves, A. DiPiazza, Sayon Roy, 2008, American Journal of Pathology.

Activation of the Acquired Immune Response Reduces Coupled Bone Formation in Response to a Periodontal Pathogen1

D. Graves, M. Siqueira, D. Faibish, 2008, The Journal of Immunology.

Diabetes causes decreased osteoclastogenesis, reduced bone formation, and enhanced apoptosis of osteoblastic cells in bacteria stimulated bone loss.

Louis C Gerstenfeld, D. Graves, L. Gerstenfeld, 2004, Endocrinology.

Diabetes Enhances Periodontal Bone Loss through Enhanced Resorption and Diminished Bone Formation

D. Graves, N. Krothapalli, Q. Luan, 2006, Journal of dental research.

Inhibition of Experimental Periodontitis by a Topical Boron-based Antimicrobial

D. Graves, T. Uchihashi, J. Plattner, 2008, Journal of dental research.

Fibroblast apoptosis induced by Porphyromonas gingivalis is stimulated by a gingipain and caspase‐independent pathway that involves apoptosis‐inducing factor

D. Graves, T. Desta, 2007, Cellular microbiology.

P. gingivalis and E. coli lipopolysaccharides exhibit different systemic but similar local induction of inflammatory markers.

D. Graves, R. Darveau, Rongkun Liu, 2008, Journal of periodontology.

Porphyromonas gingivalis and E . coli Lipopolysaccharide Exhibit Different Systemic but Similar Local Induction of Inflammatory Markers

D. Graves, R. Darveau, Rongkun Liu, 2018 .

FOXO1 Plays an Important Role in Enhanced Microvascular Cell Apoptosis and Microvascular Cell Loss in Type 1 and Type 2 Diabetic Rats

D. Graves, Sayon Roy, Y. Behl, 2009, Diabetes.

FOXO1 Plays an Important Role in Enhanced Microvascular Cell Apoptosis and Microvascular Cell Loss in Type 1 and Type 2 Diabetic Rats

D. Graves, Sayon Roy, Y. Behl, 2009, Diabetes.

Tumor Necrosis Factor Modulates Fibroblast Apoptosis, PMN Recruitment, and Osteoclast Formation in Response to P. gingivalis Infection

D. Graves, G. Naguib, Y. Jiang, 2001, Journal of dental research.

Cytokine Profiling of Macrophages Exposed to Porphyromonas gingivalis, Its Lipopolysaccharide, or Its FimA Protein

D. Graves, S. Amar, M. Fenton, 2005, Infection and Immunity.

Porphyromonas gingivalis fimbriae are pro-inflammatory but do not play a prominent role in the innate immune response to P. gingivalis

D. Graves, S. Amar, G. Naguib, 2005 .

Diabetes-enhanced tumor necrosis factor-alpha production promotes apoptosis and the loss of retinal microvascular cells in type 1 and type 2 models of diabetic retinopathy.

D. Graves, A. DiPiazza, Sayon Roy, 2008, The American journal of pathology.

Inhibition of Experimental Periodontitis by a Topical Boron-based Antimicrobial

T. Desta, D. T. Graves, Q. Luan, 2008, Journal of dental research.

Porphyromonas gingivalis and E. coli Lipopolysaccharide Exhibit Different Systemic but Similar Local Induction of Inflammatory Markers

R. Darveau, Rongkun Liu, M. Raptis, 2009 .

Porphyromonas gingivalis fimbriae are pro-inflammatory but do not play a prominent role in the innate immune response to P. gingivalis.

D. Graves, S. Amar, G. Naguib, 2005, Journal of endotoxin research.

Tumor Necrosis Factor- (cid:1) Mediates Diabetes-Enhanced Apoptosis of Matrix-Producing Cells and Impairs Diabetic Healing

Rongkun Liu, Y. Behl, T. Desta, 2006 .