H. Wen

发表

A Novel Route for F-box Protein-mediated Ubiquitination Links CHIP to Glycoprotein Quality Control*

H. Paulson, K. Glenn, Rick F. Nelson, 2006, Journal of Biological Chemistry.

Defining the Role of Ubiquitin-interacting Motifs in the Polyglutamine Disease Protein, Ataxin-3*

H. Paulson, Y. Chai, Ginger L Marrs, 2005, Journal of Biological Chemistry.

The ubiquitin ligase F-box/G-domain protein 1 promotes the degradation of the disease-linked protein torsinA through the ubiquitin–proteasome pathway and macroautophagy

H. Paulson, P. Gonzalez-Alegre, N. Bode, 2012, Neuroscience.

The ubiquitin ligase FBG1 promotes the degradation of the disease-linked protein torsinA through the ubiquitin proteasome pathway and macroautophagy

H. Paulson, P. Gonzalez-Alegre, Nicole M. Bode, 2012 .

Diversity in Tissue Expression, Substrate Binding, and SCF Complex Formation for a Lectin Family of Ubiquitin Ligases*

H. Paulson, K. Glenn, Rick F. Nelson, 2008, Journal of Biological Chemistry.

Neuronal deletion of CaV1.2 is associated with sex-specific behavioral phenotypes in mice

A. Klomp, H. Wen, Deniz A. Madencioglu, 2022, Scientific reports.

Selective Cochlear Degeneration in Mice Lacking the F-Box Protein, Fbx2, a Glycoprotein-Specific Ubiquitin Ligase Subunit

H. Paulson, T. Knutson, Richard J. H. Smith, 2007, The Journal of Neuroscience.

Deletion of the voltage‐gated calcium channel, CaV1.3, causes deficits in motor performance and associative learning

K. Parker, H. Wen, Marisol Lauffer, 2020, bioRxiv.

Using a ubiquitin ligase as an unfolded protein sensor.

K. Glenn, H. Wen, Adam Mallinger, 2012, Biochemical and biophysical research communications.

Disruption of Protein Processing in the Endoplasmic Reticulum of DYT1 Knock-in Mice Implicates Novel Pathways in Dystonia Pathogenesis

M. Ehrlich, P. Gonzalez-Alegre, N. Harata, 2016, The Journal of Neuroscience.

FBG1 Is the Final Arbitrator of A1AT-Z Degradation

K. Gibson-Corley, J. Wen, K. Glenn, 2015, PloS one.

FBG1 is a promiscuous ubiquitin ligase that sequesters APC2 and causes S-phase arrest

P. Gonzalez-Alegre, N. Kim, K. Glenn, 2010, Cell cycle.

Cellular Turnover of the Polyglutamine Disease Protein Ataxin-3 Is Regulated by Its Catalytic Activity*

H. Paulson, B. Winborn, Sokol V. Todi, 2007, Journal of Biological Chemistry.