Weichun He

发表

Melatonin activates the Nrf2‐ARE pathway when it protects against early brain injury in a subarachnoid hemorrhage model

Xue-bo Sun, Gang Chen, Huixiang Liu, 2012, Journal of pineal research.

Matrix metalloproteinase 9 may be involved in contraction of vascular smooth muscle cells in an in vitro rat model of subarachnoid hemorrhage.

Haiying Li, Haitao Shen, Weichun He, 2016, Molecular medicine reports.

Potential contribution of matrix metalloproteinase-9 (mmp-9) to cerebral vasospasm after experimental subarachnoid hemorrhage in rats.

Gang Chen, Zhong Wang, Gang Zuo, 2012, Annals of clinical and laboratory science.

Rehabilitation Treatment and Progress of Traumatic Brain Injury Dysfunction

Gang Chen, Wenli Chen, Weichun He, 2017, Neural plasticity.

Potential Contribution of Hypoxia-Inducible Factor-1α, Aquaporin-4, and Matrix Metalloproteinase-9 to Blood–Brain Barrier Disruption and Brain Edema After Experimental Subarachnoid Hemorrhage

Xue-bo Sun, Gang Chen, Huixiang Liu, 2012, Journal of Molecular Neuroscience.

Cyclophilin A/Cluster of Differentiation 147 Interactions Participate in Early Brain Injury After Subarachnoid Hemorrhage in Rats

Haiying Li, Haitao Shen, Gang Chen, 2015, Critical care medicine.

A Therapeutic Target of Cerebral Hemorrhagic Stroke: Matrix Metalloproteinase- 9.

Gang Chen, Xiaochun Duan, Zhong Wang, 2017, Current drug targets.

Melatonin activates the Nrf2‐ARE pathway when it protects against early brain injury in a subarachnoid hemorrhage model

Xue-bo Sun, Gang Chen, Huixiang Liu, 2012, Journal of pineal research.

Cyclophilin A/Cluster of Differentiation 147 Interactions Participate in Early Brain Injury After Subarachnoid Hemorrhage in Rats

Haiying Li, Haitao Shen, Gang Chen, 2015, Critical care medicine.

Potential Contribution of Hypoxia-Inducible Factor-1α, Aquaporin-4, and Matrix Metalloproteinase-9 to Blood–Brain Barrier Disruption and Brain Edema After Experimental Subarachnoid Hemorrhage

Xue-bo Sun, Gang Chen, Huixiang Liu, 2012, Journal of Molecular Neuroscience.