L. Orejana

Sildenafil ameliorates cognitive deficits and tau pathology in a senescence-accelerated mouse model

J. Jordán, E. Puerta, N. Aguirre, 2012, Neurobiology of Aging.

Propranolol reduces cognitive deficits, amyloid β levels, tau phosphorylation and insulin resistance in response to chronic corticosterone administration.

M. Ramírez, Marta Dobarro, Lourdes Orejana, 2013, The international journal of neuropsychopharmacology.

Modulation of the ASK1–MKK3/6–p38/MAPK signalling pathway mediates sildenafil protection against chemical hypoxia caused by malonate

E. Puerta, B. Goñi-Allo, N. Aguirre, 2013, British journal of pharmacology.

Sildenafil Decreases BACE1 and Cathepsin B Levels and Reduces APP Amyloidogenic Processing in the SAMP8 Mouse.

J. Jordán, A. Cedazo-Mínguez, E. Puerta, 2015, The journals of gerontology. Series A, Biological sciences and medical sciences.

Propranolol restores cognitive deficits and improves amyloid and Tau pathologies in a senescence-accelerated mouse model

M. Ramírez, N. Aguirre, L. Orejana, 2013, Neuropharmacology.

Long‐lasting neuroprotective effect of sildenafil against 3,4‐methylenedioxymethamphetamine‐ induced 5‐hydroxytryptamine deficits in the rat brain

R. de la Torre, J. Jordán, E. Puerta, 2012, Journal of neuroscience research.

Inhibition of calpain-regulated p35/cdk5 plays a central role in sildenafil-induced protection against chemical hypoxia produced by malonate.

J. Jordán, Manuela G. López, E. Puerta, 2013, Biochimica et biophysica acta.

Implication of JNK pathway on tau pathology and cognitive decline in a senescence-accelerated mouse model

E. Puerta, N. Aguirre, L. Orejana, 2013, Experimental Gerontology.

Propranolol reduces cognitive deficits, amyloid β levels, tau phosphorylation and insulin resistance in response to chronic corticosterone administration.

M. Ramírez, N. Aguirre, L. Orejana, 2013, The international journal of neuropsychopharmacology.