E. Boettger

The mitochondrial ribosome of the protozoan Acanthamoeba castellanii is the target for macrolide antibiotics.

P. Wild, A. Mathis, P. Deplazes, 2004, Molecular and biochemical parasitology.

Oxidative stress response genes in Mycobacterium tuberculosis: role of ahpC in resistance to peroxynitrite and stage-specific survival in macrophages.

V. Deretic, B. Springer, P. Sander, 2002, Microbiology.

High-level resistance to isoniazid and ethionamide in multidrug-resistant Mycobacterium tuberculosis of the Lisboa family is associated with inhA double mutations.

M. Viveiros, I. Couto, J. Ramos, 2013, The Journal of antimicrobial chemotherapy.

Mitochondrial Ribosome as the Target for the Macrolide Antibiotic Clarithromycin in the Helminth Echinococcus multilocularis

C. Kapel, P. Wild, A. Mathis, 2005, Antimicrobial Agents and Chemotherapy.

Clinical implications of molecular drug resistance testing for Mycobacterium tuberculosis: a TBNET/RESIST-TB consensus statement.

S Niemann, F. Cobelens, F. van Leth, 2016, The international journal of tuberculosis and lung disease : the official journal of the International Union against Tuberculosis and Lung Disease.

Mitochondrial misreading in skeletal muscle accelerates metabolic aging and confers lipid accumulation and increased inflammation

S. Frank, E. Laczko, D. Shcherbakov, 2020, RNA.

Silencing of Oxidative Stress Response in Mycobacterium tuberculosis: Expression Patterns of ahpC in Virulent and Avirulent Strains and Effect ofahpC Inactivation

V. Deretic, K. Papavinasasundaram, B. Springer, 2001, Infection and Immunity.

Silencing of Oxidative Stress Response in Mycobacterium tuberculosis: Expression Patterns of ahpC in Virulent and Avirulent Strains and Effect ofahpC Inactivation

V. Deretic, K. Papavinasasundaram, M. Colston, 2001, Infection and Immunity.