K. Rogulski

发表

Efficacy and toxicity of replication-competent adenovirus-mediated double suicide gene therapy in combination with radiation therapy in an orthotopic mouse prostate cancer model.

J. H. Kim, K. Barton, S. Freytag, 2002, International journal of radiation oncology, biology, physics.

Evaluation of the biodistribution, persistence, toxicity, and potential of germ-line transmission of a replication-competent human adenovirus following intraprostatic administration in the mouse.

J. H. Kim, S. Freytag, A. Kolozsvary, 2000, Molecular therapy : the journal of the American Society of Gene Therapy.

Advances in Brief In Vivo Antitumor Activity of ONYX-015 Is Influenced by p 53 Status and Is Augmented by Radiotherapy 1

J. H. Kim, D. Kirn, S. Freytag, 2000 .

In vivo antitumor activity of ONYX-015 is influenced by p53 status and is augmented by radiotherapy.

D. Kirn, J. H. Kim, S. Freytag, 2000, Cancer research.

Glioma cells transduced with an Escherichia coli CD/HSV-1 TK fusion gene exhibit enhanced metabolic suicide and radiosensitivity.

S. Kim, J. Kim, S. Freytag, 1997, Human gene therapy.

A novel three-pronged approach to kill cancer cells selectively: concomitant viral, double suicide gene, and radiotherapy.

J. H. Kim, S. Freytag, D. Paielli, 1998, Human gene therapy.

Double suicide gene therapy augments the antitumor activity of a replication-competent lytic adenovirus through enhanced cytotoxicity and radiosensitization.

J. H. Kim, S. Freytag, M. Wing, 2000, Human gene therapy.

Pronounced antitumor effects and tumor radiosensitization of double suicide gene therapy.

S. Freytag, A. Kolozsvary, K. Rogulski, 1997, Clinical cancer research : an official journal of the American Association for Cancer Research.

The Negative c-Myc Target Onzin Affects Proliferation and Apoptosis via Its Obligate Interaction with Phospholipid Scramblase I

E. Prochownik, P. Sims, Quansheng Zhou, 2006, Molecular and Cellular Biology.

Onzin, a c-Myc-repressed target, promotes survival and transformation by modulating the Akt–Mdm2–p53 pathway

Simon C Watkins, E. Prochownik, K. Rothermund, 2005, Oncogene.

C-Myc-independent restoration of multiple phenotypes by two C-Myc target genes with overlapping functions.

E. Prochownik, J. Sedivy, A. Whiting, 2005, Cancer research.

Myc Target in Myeloid Cells-1, a Novel c-Myc Target, Recapitulates Multiple c-Myc Phenotypes*

E. Prochownik, Xiaoying Yin, L. Grove, 2002, The Journal of Biological Chemistry.

Multiple interacting elements delineate an ecdysone-dependent regulatory region with secondary responsive character.

I. L. Cartwright, K. Rogulski, 1995, Journal of molecular biology.

Deregulation of common genes by c-Myc and its direct target, MT-MC1.

D. Corcoran, P. Benos, E. Prochownik, 2005, Proceedings of the National Academy of Sciences of the United States of America.

Selective radiosensitization of 9L glioma in the brain transduced with double suicide fusion gene.

S. Freytag, A. Kolozsvary, M. Khil, 1998, The cancer journal from Scientific American.