A. Richardson

发表

Necroptosis increases with age in the brain and contributes to age-related neuroinflammation

Tommy L. Lewis, S. S. Deepa, Sabira Mohammed, 2021, GeroScience.

alteration in and in the

W. Freeman, Dustin R Masser, N. Hadad, 2017 .

Formation of 3‐nitrotyrosines in carbonic anhydrase III is a sensitive marker of oxidative stress in skeletal muscle

J. Faulkner, R. Beynon, D. Simpson, 2007, Proteomics. Clinical applications.

Deletion of Neuronal CuZnSOD Accelerates Age-Associated Muscle Mitochondria and Calcium Handling Dysfunction That Is Independent of Denervation and Precedes Sarcopenia

H. Van Remmen, D. Claflin, S. Brooks, 2021, International journal of molecular sciences.

Removal of p16 INK4 Expressing Cells in Late Life has Moderate Beneficial Effects on Skeletal Muscle Function in Male Mice

J. Markworth, A. McArdle, H. Van Remmen, 2022, Frontiers in Aging.

Senolytic treatment reduces cell senescence and necroptosis in Sod1 knockout mice that is associated with reduced inflammation and hepatocellular carcinoma

H. Van Remmen, S. S. Deepa, Sabira Mohammed, 2022, bioRxiv.

W. Freeman, Dustin R Masser, N. Hadad, 2017 .

Scavenging mitochondrial hydrogen peroxide by peroxiredoxin 3 overexpression attenuates contractile dysfunction and muscle atrophy in a murine model of accelerated sarcopenia

W. Freeman, M. Kinter, H. Van Remmen, 2022, Aging cell.

Neuronal deletion of MnSOD in mice leads to demyelination, inflammation and progressive paralysis that mimics phenotypes associated with progressive multiple sclerosis

Tommy L. Lewis, S. Plafker, N. Smith, 2022, Redox biology.

Analysis of the transcriptional activity of the 5′‐flanking region of the rat catalase gene in transiently transfected cells and in transgenic mice

H. Van Remmen, C. Walter, A. Richardson, 1998, Journal of cellular physiology.

Increased Oxidative Damage Is Correlated to Altered Mitochondrial Function in Heterozygous Manganese Superoxide Dismutase Knockout Mice*

C. Epstein, H. Van Remmen, C. C. Conrad, 1998, The Journal of Biological Chemistry.

Changes in Expression of Antioxidant Enzymes Affect Cell-Mediated LDL Oxidation and Oxidized LDL-Induced Apoptosis in Mouse Aortic Cells

C. Epstein, J. Vijg, Y. Ho, 2001, Arteriosclerosis, thrombosis, and vascular biology.

Superoxide-mediated oxidative stress accelerates skeletal muscle atrophy by synchronous activation of proteolytic systems

Jeongmoon J. Choi, Jeongmoon J Choi, Young C. Jang, 2020, GeroScience.

Molecular changes in transcription and metabolic pathways underlying muscle atrophy in the CuZnSOD null mouse model of sarcopenia

Caroline S. Kinter, W. Freeman, J. Wren, 2020, GeroScience.

The Geropathology Grading Platform demonstrates that mice null for Cu/Zn-superoxide dismutase show accelerated biological aging

J. Stoner, T. Snider, S. S. Deepa, 2018, GeroScience.

Free radical generation by skeletal muscle of adult and old mice: effect of contractile activity

J. Faulkner, H. Remmen, M. Jackson, 2006, Aging cell.

Deletion of Sod1 in motor neurons exacerbates age-related changes in axons and NMJs associated with premature muscle atrophy in aging mice

A. McArdle, H. Van Remmen, A. Vasilaki, 2022, bioRxiv.

Expression of genes coding for antioxidant enzymes and heat shock proteins is altered in primary cultures of rat hepatocytes

H. Chung, B. Yu, A. Richardson, 1996, Journal of cellular physiology.

A reliable assessment of 8-oxo-2-deoxyguanosine levels in nuclear and mitochondrial DNA using the sodium iodide method to isolate DNA.

I. Thompson, S. Austad, D. Troyer, 2001, Nucleic acids research.

Characterization of the antioxidant status of the heterozygous manganese superoxide dismutase knockout mouse.

C. Epstein, H. Remmen, H. Yang, 1999, Archives of biochemistry and biophysics.