S. K. Bremner

Metformin suppresses adipogenesis through both AMP-activated protein kinase (AMPK)-dependent and AMPK-independent mechanisms

B. Viollet, S. Ahmed, S. Yarwood, 2016, Molecular and Cellular Endocrinology.

Pleiotropic effects of Syntaxin16 identified by gene editing in cultured adipocytes

B. Hudson, C. Sutherland, G. Gould, 2022, Frontiers in Cell and Developmental Biology.

A systematic analysis of anti-diabetic medicinal plants from cells to clinical trials

G. Gould, S. Omale, T. Johnson, 2023, PeerJ.

Corrigendum to “Metformin suppresses adipogenesis through both AMP-activated protein kinase (AMPK)-dependent and AMPK-independent mechanisms” [Mol. Cell. Endocrinol. 440 15 January 2017 57–68]

B. Viollet, S. Yarwood, J. Dunlop, 2017, Molecular and Cellular Endocrinology.

Phosphorylation of the N-terminus of Syntaxin-16 controls interaction with mVps45 and GLUT4 trafficking in adipocytes

C. Sutherland, G. Gould, N. J. Bryant, 2023, PeerJ.

Metformin suppresses adipogenesis through both AMP-activated protein kinase (AMPK)-dependent and AMPK-independent mechanisms

J. Dunlop, I. Salt, P. J. Logan, 2017, Molecular and Cellular Endocrinology.

EHD2 regulates plasma membrane integrity and downstream insulin receptor signaling events

H. Ardalani, Franziska Kopietz, S. K. Bremner, 2023, Molecular biology of the cell.

The Anti-diabetic Drug, Metformin, Suppresses Adipogenesis through both AMP-activated Protein Kinase (AMPK)-dependent and AMPK-independent Mechanisms

B. Viollet, S. Yarwood, J. Dunlop, 2016 .