Hiromichi Murase

Does glycemic control reverse dispersion of ventricular repolarization in type 2 diabetes?

T. Miura, H. Akasaka, S. Saitoh, 2014, Cardiovascular Diabetology.

Translocation of GSK-3β, a trigger of permeability transition, is kinase activity-dependent and mediated by interaction with VDAC2

Y. Horio, S. Ishikawa, T. Miura, 2014 .

Role of connexin-43 in protective PI3K-Akt-GSK-3β signaling in cardiomyocytes.

S. Ishikawa, T. Miura, Tatsuya Sato, 2012, American journal of physiology. Heart and circulatory physiology.

Translocation of Glycogen Synthase Kinase-3β (GSK-3β), a Trigger of Permeability Transition, Is Kinase Activity-dependent and Mediated by Interaction with Voltage-dependent Anion Channel 2 (VDAC2)*

Y. Horio, S. Ishikawa, T. Miura, 2014, The Journal of Biological Chemistry.

Cytoprotective regulation of the mitochondrial permeability transition pore is impaired in type 2 diabetic Goto-Kakizaki rat hearts.

T. Miura, Tatsuya Sato, A. Kuno, 2012, Journal of molecular and cellular cardiology.

Diabetes increases the susceptibility to acute kidney injury after myocardial infarction through augmented activation of renal Toll-like receptors in rats.

S. Ishikawa, T. Miura, A. Kuno, 2017, American journal of physiology. Heart and circulatory physiology.

Inhibition of DPP-4 reduces acute mortality after myocardial infarction with restoration of autophagic response in type 2 diabetic rats

S. Ishikawa, T. Miura, A. Kuno, 2015, Cardiovascular Diabetology.

Excessive degradation of adenine nucleotides by up-regulated AMP deaminase underlies afterload-induced diastolic dysfunction in the type 2 diabetic heart.

S. Ishikawa, T. Miura, Tatsuya Sato, 2015, Journal of molecular and cellular cardiology.