L. C. Souza

Expression of inducible nitric oxide synthase is increased in patients with heart failure due to ischemic disease.

C. Scavone, P. D. da Luz, A. Chagas, 2004, Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas.

Oxidized thiols markedly amplify the vascular response to balloon injury in rabbits through a redox active metal-dependent pathway.

C. Pasqualucci, M. Janiszewski, F. Laurindo, 1998, Cardiovascular research.

Alamandine enhances cardiomyocyte contractility in hypertensive rats through a NO-dependent activation of CaMKII.

S. Guatimosim, A. Santos, M. Campagnole-Santos, 2020, American journal of physiology. Cell physiology.

Inhibition of vascular NADH/NADPH oxidase activity by thiol reagents: lack of correlation with cellular glutathione redox status.

M. Janiszewski, M. Pedro, F. Laurindo, 2000, Free radical biology & medicine.

Vascular oxidant stress early after balloon injury: evidence for increased NAD(P)H oxidoreductase activity.

J. Krieger, F. Laurindo, M. L. Junqueira, 2000, Free radical biology & medicine.

Oxidative stress as a signaling mechanism of the vascular response to injury: the redox hypothesis of restenosis.

L. Azevedo, M. Janiszewski, M. Pedro, 2000, Cardiovascular research.

Cyclo‐Gly‐Pro, a cyclic dipeptide, attenuates nociceptive behaviour and inflammatory response in mice

B. Diaz, L. Quintans-Júnior, J. Ferro, 2015, Clinical and experimental pharmacology & physiology.