A. Ståhl

Resistance against ethacrynic acid in glutathione transferase 7-7 (GST-P)-positive hepatocytes isolated from carcinogen-treated rats: the role of cytoskeletal changes and ATP depletion.

U. Stenius, J. Högberg, M. Warholm, 1995, Toxicology in vitro : an international journal published in association with BIBRA.

Lipid peroxidation in vivo monitored as ethane exhalation and malondialdehyde excretion in urine after oral administration of chloroform.

J. Högberg, A. Ståhl, K. Sigvardsson, 2009, Acta pharmacologica et toxicologica.

In vitro studies on non-genotoxic carcinogens: resistance to DNA synthesis inhibition in GST-P-positive hepatocytes isolated from enzyme-altered foci-bearing rats.

U. Stenius, J. Högberg, A. Ståhl, 1998, Toxicology in vitro : an international journal published in association with BIBRA.

Absorption of selenite in the rat small intestine: interactions with glutathione.

J. Högberg, I. Anundi, A. Ståhl, 2009, Acta pharmacologica et toxicologica.

Involvement of glutathione reductase in selenite metabolism and toxicity, studied in isolated rat hepatocytes

J. Högberg, I. Anundi, A. Ståhl, 1982, Archives of Toxicology.

Studies of the role of DNA fragmentation in selenium toxicity.

J. Högberg, M. Warholm, A. Ståhl, 1988, Biochemical pharmacology.

Effects of selenite on O2 consumption, glutathione oxidation and NADPH levels in isolated hepatocytes and the role of redox changes in selenite toxicity.

J. Högberg, I. Anundi, A. Ståhl, 1984, Chemico-biological interactions.

Reduced ATM kinase activity and an attenuated p53 response to DNA damage in carcinogen-induced preneoplastic hepatic lesions in the rat.

Ilona Silins, U. Stenius, J. Högberg, 2001, Carcinogenesis.

Selenite biotransformation to volatile metabolites in an isolated hepatocyte model system.

J. Högberg, I. Anundi, A. Ståhl, 1984, Biochemical pharmacology.

Studies on Selenite-Induced DNA Fragmentation and the Role of Poly(ADP Ribose)Polymerase in Selenite Toxicity

J. Högberg, A. Ståhl, P. Garberg, 1989 .