Sylvia Torres-Odio

发表

Loss of mitochondrial ClpP, Lonp1, and Tfam triggers transcriptional induction of Rnf213, a susceptibility factor for moyamoya disease

C. López-Otín, I. Wittig, A. West, 2020, neurogenetics.

Increased presence of nuclear DNAJA3 and upregulation of cytosolic STAT1 and of nucleic acid sensors trigger innate immunity in the ClpP-null mouse

I. Wittig, A. West, G. Auburger, 2021, neurogenetics.

Prominent Increases of Nuclear DNAJA3and Cytosolic STAT1 with Nucleic Acid Sensors Underlie Innate Immunity Activation in ClpP-null Mouse

A. West, G. Auburger, S. Gispert, 2021 .

Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation

D. Meierhofer, M. Mittelbronn, P. Harter, 2017, Journal of Neuroinflammation.

Inflammatory profile in a canine model of hypothermic circulatory arrest.

W. Baumgartner, M. Johnston, E. Etchill, 2021, The Journal of surgical research.

Borrelia burgdorferi engages mammalian type I interferon responses via the cGAS-STING pathway

A. West, L. G. Adams, Lauren C. Farris, 2022, bioRxiv.

The Splicing Factor hnRNP M Is a Critical Regulator of Innate Immune Gene Expression in Macrophages

K. Patrick, A. West, A. West, 2019, bioRxiv.

Stress-induced dynamic regulation of mitochondrial STAT3 and its association with cyclophilin D reduce mitochondrial ROS production

P. Shaw, H. Baumann, K. Takabe, 2017, Science Signaling.

Elevated type I interferon responses potentiate metabolic dysfunction, inflammation, and accelerated aging in mtDNA mutator mice

A. West, S. Bell, R. Watson, 2021, Science Advances.

Loss of Mitochondrial Protease CLPP Activates Type I IFN Responses through the Mitochondrial DNA–cGAS–STING Signaling Axis

I. Wittig, A. West, G. Auburger, 2021, The Journal of Immunology.

Type I interferon potentiates metabolic dysfunction, inflammation, and accelerated aging in mtDNA mutator mice

A. West, C. Martinez, S. Bell, 2020, bioRxiv.

Impaired lysosomal acidification triggers iron deficiency and inflammation in vivo

J. Frahm, Takashi Watanabe, A. West, 2019, eLife.

Impaired lysosomal acidification triggers iron deficiency, necrotic cell death and inflammation in vivo

J. Frahm, Takashi Watanabe, E. Meyron-Holtz, 2019, bioRxiv.

Search for SCA2 blood RNA biomarkers highlights Ataxin-2 as strong modifier of the mitochondrial factor PINK1 levels

G. Akdal, G. Auburger, S. Gispert, 2016, Neurobiology of Disease.

CLPP Depletion Causes Diplotene Arrest; Underlying Testis Mitochondrial Dysfunction Occurs with Accumulation of Perrault Proteins ERAL1, PEO1, and HARS2

A. West, W. Baarends, C. Münch, 2022, Cells.

Inactivity of Peptidase ClpP Causes Primary Accumulation of Mitochondrial Disaggregase ClpX with Its Interacting Nucleoid Proteins, and of mtDNA

H. Prokisch, S. Sieber, I. Wittig, 2021, Cells.

Inactivity of Peptidase ClpP Causes Primary Accumulation of Mitochondrial Disaggregase ClpX With Its Interactors at the Nucleoid and RNA Granule

H. Prokisch, W. Newman, S. Sieber, 2021 .

SerThr-PhosphoProteome of Brain from Aged PINK1-KO+A53T-SNCA Mice Reveals pT1928-MAP1B and pS3781-ANK2 Deficits, as Hub between Autophagy and Synapse Changes

M. Jendrach, G. Auburger, S. Gispert, 2019, International journal of molecular sciences.

Loss of mitochondrial protease CLPP activates type I interferon responses through the mtDNA-cGAS-STING signaling axis

I. Wittig, A. West, G. Auburger, 2020 .

Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation

D. Meierhofer, M. Mittelbronn, P. Harter, 2017, Journal of Neuroinflammation.

TRIM14 Is a Key Regulator of the Type I IFN Response during Mycobacterium tuberculosis Infection

K. Patrick, A. West, J. Cox, 2020, The Journal of Immunology.