S. Marsh

发表

Cardiovascular dysfunction in Zucker obese and Zucker diabetic fatty rats: role of hydronephrosis.

L. Dell’Italia, J. Chatham, A. Agarwal, 2007, American journal of physiology. Heart and circulatory physiology.

Evidence for distinct effects of exercise in different cardiac hypertrophic disorders.

B. Dieter, Emily J Johnson, S. Marsh, 2015, Life sciences.

Immediate effects of a single exercise bout on protein O-GlcNAcylation and chromatin regulation of cardiac hypertrophy.

K. Porter, S. Marsh, H. Medford, 2013, American journal of physiology. Heart and circulatory physiology.

Glutathione depletion and acute exercise increase O-GlcNAc protein modification in rat skeletal muscle

V. Dalbo, D. Briskey, P. Tucker, 2014, Molecular and Cellular Biochemistry.

Inhibition of O‐GlcNAcase in perfused rat hearts by NAG‐thiazolines at the time of reperfusion is cardioprotective in an O‐GlcNAc dependent manner

R. Marchase, J. Chatham, B. Laczy, 2009, American journal of physiology. Heart and circulatory physiology.

A Systematic Review of Fetal Genes as Biomarkers of Cardiac Hypertrophy in Rodent Models of Diabetes

S. Marsh, E. Cox, 2014, PloS one.

Protein O-GlcNAcylation and Cardiovascular (Patho)physiology*

J. Chatham, Helen E Collins, S. Marsh, 2014, The Journal of Biological Chemistry.

Evidence for a non-antioxidant, dose-dependent role of α -lipoic acid in caspase-3 and ERK2 activation in endothelial cells

G. Gobe, J. Coombes, B. Pat, 2005, Apoptosis.

Cardiac O-GlcNAcylation blunts autophagic signaling in the diabetic heart.

L. Dell’Italia, J. Chatham, P. Powell, 2013, Life sciences.

Exercise and the endothelial cell.

J. Coombes, S. Marsh, 2005, International journal of cardiology.

Consuming a Western diet for two weeks suppresses fetal genes in mouse hearts.

Lindsey Miller, S. Marsh, E. Cox, 2014, American journal of physiology. Regulatory, integrative and comparative physiology.

High-fat, low-carbohydrate diet promotes arrhythmic death and increases myocardial ischemia-reperfusion injury in rats.

L. Dell’Italia, S. Lloyd, S. Litovsky, 2014, American journal of physiology. Heart and circulatory physiology.

The role of O-GlcNAc transferase in regulating the gene transcription of developing and failing hearts.

S. Marsh, H. Medford, 2014, Future cardiology.

The paradoxical world of protein O-GlcNAcylation: a novel effector of cardiovascular (dys)function.

J. Chatham, S. Marsh, 2011, Cardiovascular research.

Effects of exercise training and RhoA/ROCK inhibition on plaque in ApoE-/- mice.

J. D. de Haan, R. Fassett, J. Coombes, 2013, International journal of cardiology.

Antioxidant supplementation reduces skeletal muscle mitochondrial biogenesis.

G. Wadley, J. Peake, J. Coombes, 2011, Medicine and science in sports and exercise.

Bcl-2 in endothelial cells is increased by vitamin E and alpha-lipoic acid supplementation but not exercise training.

P. Laursen, G. Gobe, J. Coombes, 2005, Journal of molecular and cellular cardiology.

Interaction of diet and diabetes on cardiovascular function in rats.

L. Dell’Italia, J. Chatham, S. Marsh, 2009, American journal of physiology. Heart and circulatory physiology.

Post-translational protein modification by O-linked N-acetyl-glucosamine: its role in mediating the adverse effects of diabetes on the heart.

J. Chatham, S. Marsh, Jennifer L Mclarty, 2013, Life sciences.

Alpha-lipoic acid does not acutely affect resistance and conduit artery function or oxidative stress in healthy men.

J. Sharman, J. Coombes, J. Cockcroft, 2004, British journal of clinical pharmacology.

Dietary Supplementation of Vitamin E and α-Lipoic Acid Upregulates Cell Growth and Signaling Genes in Rat Myocardium

J. Coombes, S. A. Marsh, S. Mason, 2006, International journal of biomedical science : IJBS.

Cardioprotection requires flipping the 'posttranslational modification' switch.

K. Porter, S. Marsh, H. Medford, 2012, Life sciences.

Vitamin E and a-Lipoic Acid Supplementation Increase Bleeding Tendency via an Intrinsic Coagulation Pathway

J. Coombes, S. Marsh, 2006, Clinical and applied thrombosis/hemostasis : official journal of the International Academy of Clinical and Applied Thrombosis/Hemostasis.

Exercise and diabetes have opposite effects on the assembly and O-GlcNAc modification of the mSin3A/HDAC1/2 complex in the heart

S. A. Marsh, E. J. Cox, S. Marsh, 2013, Cardiovascular Diabetology.

Oxidative stress biomarkers as predictors of cardiovascular disease.

J. Coombes, R. Fassett, S. Marsh, 2011, International journal of cardiology.

Effects of exercise and antioxidant supplementation on endothelial gene expression.

J. D. de Haan, S. Mason, L. Ward, 2012, International journal of cardiology.

O-GlcNAc protein modification in C2C12 myoblasts exposed to oxidative stress indicates parallels with endogenous antioxidant defense.

S. A. Marsh, V. Dalbo, P. Tucker, 2015, Biochemistry and cell biology = Biochimie et biologie cellulaire.

Altering the redox state of skeletal muscle by glutathione depletion increases the exercise‐activation of PGC‐1α

G. Wadley, J. Peake, D. Briskey, 2014, Physiological reports.

Activation of the hexosamine biosynthesis pathway and protein O-GlcNAcylation modulate hypertrophic and cell signaling pathways in cardiomyocytes from diabetic mice

Louis J. Dell’Italia, L. Dell’Italia, J. Chatham, 2011, Amino Acids.

Chronic ingestion of a Western diet increases O-linked-β-N-acetylglucosamine (O-GlcNAc) protein modification in the rat heart.

J. Chatham, S. Marsh, H. Medford, 2012, Life sciences.

Physiological Responses to the Menstrual Cycle Implications for the Development of Heat Illness in Female Athletes

S. A. Marsh, D. Jenkins, S. Marsh, 2002 .

Physiological Responses to the Menstrual Cycle

D. Jenkins, S. Marsh, 2002, Sports medicine.

Manipulating training intensity and volume in already well-trained rats: effect on skeletal muscle oxidative and glycolytic enzymes and buffering capacity.

P. Laursen, D. Jenkins, J. Coombes, 2007, Applied physiology, nutrition, and metabolism = Physiologie appliquee, nutrition et metabolisme.

Modification of STIM1 by O-linked N-Acetylglucosamine (O-GlcNAc) Attenuates Store-operated Calcium Entry in Neonatal Cardiomyocytes*

R. Marchase, J. Chatham, Luyun Zou, 2012, The Journal of Biological Chemistry.

Glutathione depletion and acute exercise increase O-GlcNAc protein modification in rat skeletal muscle

V. Dalbo, D. Briskey, P. Tucker, 2015, Molecular and Cellular Biochemistry.

Effects of Antioxidant Supplementation and Exercise Training on Erythrocyte Antioxidant Enzymes

P. Laursen, J. Coombes, S. Marsh, 2006 .

A novel protocol for assessing exercise performance and dystropathophysiology in the mdx mouse

B. D. Rodgers, J. A. Eldridge, S. Marsh, 2014, Muscle & nerve.

Protein O-GlcNAcylation and Cardiovascular (Patho)physiology*

J. Chatham, S. Marsh, Helen E. Collins, 2014, The Journal of Biological Chemistry.

The role of O-GlcNAc transferase in regulating the gene transcription of developing and failing hearts.

S. Marsh, H. Medford, 2014, Future cardiology.

Cardiac O-GlcNAcylation blunts autophagic signaling in the diabetic heart.

L. Dell’Italia, J. Chatham, P. Powell, 2013, Life sciences.