M. Puklo

Lack of cathelicidin processing in Papillon-Lefèvre syndrome patients reveals essential role of LL-37 in periodontal homeostasis

J. Potempa, P. Eickholz, P. Hiemstra, 2014, Orphanet Journal of Rare Diseases.

Analysis of neutrophil-derived antimicrobial peptides in gingival crevicular fluid suggests importance of cathelicidin LL-37 in the innate immune response against periodontogenic bacteria.

J. Potempa, P. Hiemstra, S. Eick, 2008, Oral microbiology and immunology.

Neutrophils in chronic and aggressive periodontitis in interaction with Porphyromonas gingivalis and Aggregatibacter actinomycetemcomitans.

J. Potempa, S. Eick, W. Pfister, 2009, Journal of periodontal research.

Functional Cathepsin C mutations cause different Papillon-Lefèvre syndrome phenotypes.

H. Schackert, P. Eickholz, T. Hoffmann, 2008, Journal of clinical periodontology.

Influence of serum on interaction of Porphyromonas gingivalis ATCC 33277 and Aggregatibacter actinomycetemcomitans Y4 with an epithelial cell line.

S. Eick, W. Pfister, P. Preshaw, 2010, Journal of periodontal research.

Triggering NETosis via protease-activated receptor (PAR)-2 signaling as a mechanism of hijacking neutrophils function for pathogen benefits

J. Potempa, M. Lech, S. Eick, 2019, PLoS pathogens.

Degradation of Human Antimicrobial Peptide LL-37 by Staphylococcus aureus-Derived Proteinases

W. Shafer, B. Lupa, T. Foster, 2004, Antimicrobial Agents and Chemotherapy.

Variables of host response in saliva of patients with periodontitis: a pilot study.

Arndt Guentsch, Michael Kiehntopf, Wolfgang Pfister, 2012, Quintessence international.