Esther Barth

发表

CLUH controls astrin-1 expression to couple mitochondrial metabolism to cell cycle progression

E. Rugarli, T. Langer, P. Giavalisco, 2021, bioRxiv.

Astrocyte‐specific deletion of the mitochondrial m‐AAA protease reveals glial contribution to neurodegeneration

E. Rugarli, P. Kloppenburg, T. Langer, 2019, Glia.

Modulation of Proteolytic Processing by Glycosphingolipids Generates Amyloid β-Peptide

J. Walter, K. Sandhoff, I. Tamboli, 2006 .

Alternative Splicing of Spg7, a Gene Involved in Hereditary Spastic Paraplegia, Encodes a Variant of Paraplegin Targeted to the Endoplasmic Reticulum

E. Rugarli, G. Mancuso, P. Crivello, 2012, PloS one.

Profile of cholesterol-related sterols in aged amyloid precursor protein transgenic mouse brain DOI 10.1194/jlr.M200071-JLR200

T. Bayer, M. Staufenbiel, G. Multhaup, 2002, Journal of Lipid Research.

Loss of the mitochondrial i‐AAA protease YME1L leads to ocular dysfunction and spinal axonopathy

E. Rugarli, T. Wai, T. Langer, 2018, EMBO molecular medicine.

Statins Promote the Degradation of Extracellular Amyloid β-Peptide by Microglia via Stimulation of Exosome-associated Insulin-degrading Enzyme (IDE) Secretion*

M. Siepmann, M. Heneka, J. Walter, 2010, The Journal of Biological Chemistry.

CLUH regulates mitochondrial biogenesis by binding mRNAs of nuclear-encoded mitochondrial proteins

E. Rugarli, P. Frommolt, M. Sardiello, 2014, The Journal of cell biology.

JCB_201403129 1..11

E. Rugarli, P. Frommolt, M. Sardiello, 2014 .

The class 3 PI3K coordinates autophagy and mitochondrial lipid catabolism by controlling nuclear receptor PPARα

E. Rugarli, H. Guillou, M. Pende, 2019, Nature Communications.

CLUH regulates mitochondrial metabolism by controlling translation and decay of target mRNAs

E. Rugarli, V. Kondylis, V. Velagapudi, 2017, The Journal of cell biology.

The Mitochondrial m-AAA Protease Prevents Demyelination and Hair Greying

E. Rugarli, T. Langer, P. Martinelli, 2016, PLoS genetics.

The class 3 PI3K coordinates autophagy and mitochondrial lipid catabolism by controlling nuclear receptor PPARα

E. Rugarli, H. Guillou, M. Pende, 2019, Nature Communications.

Inhibition of Glycosphingolipid Biosynthesis Reduces Secretion of the β-Amyloid Precursor Protein and Amyloid β-Peptide*[boxs]

M. Heneka, J. Walter, K. Sandhoff, 2005, Journal of Biological Chemistry.

Inhibition of glycosphingolipid biosynthesis reduces secretion of the beta-amyloid precursor protein and amyloid beta-peptide.

M. Heneka, J. Walter, K. Sandhoff, 2005, The Journal of biological chemistry.

AFG3L2 supports mitochondrial protein synthesis and Purkinje cell survival.

E. Rugarli, P. Kloppenburg, T. Langer, 2012, The Journal of clinical investigation.

CLUH controls astrin-1 expression to couple mitochondrial metabolism to cell cycle progression

E. Rugarli, T. Langer, P. Giavalisco, 2021, bioRxiv.

A Structural Switch of Presenilin 1 by Glycogen Synthase Kinase 3β-mediated Phosphorylation Regulates the Interaction with β-Catenin and Its Nuclear Signaling*

C. Haass, J. Walter, R. Fluhrer, 2007, Journal of Biological Chemistry.

CLUH maintains functional mitochondria and translation in motoneuronal axons and prevents peripheral neuropathy

H. Lehmann, Esther Barth, Jean-Michel Cioni, 2024, Science advances.