M. Nur-e-Kamal

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Differential Requirement for Rho Family GTPases in an Oncogenic Insulin-like Growth Factor-I Receptor-induced Cell Transformation*

Weiqun Li, Lu-Hai Wang, T. Miki, 2001, The Journal of Biological Chemistry.

The role of Gln61 and Glu63 of Ras GTPases in their activation by NF1 and Ras GAP.

H. Maruta, M. Nur-e-Kamal, 1992, Molecular biology of the cell.

Targeted delivery of human neurofibromin and c-Raf-1 mutants to the cytoplasmic membrane by use of the influenza virus hemagglutinin.

A. Herrmann, E. Ponimaskin, M. Schmidt, 1997, Biochimica et biophysica acta.

Midkine (MK), a retinoic acid (RA)-inducible gene product, produced in E. coli acts on neuronal and HL60 leukemia cells.

A. Burgess, L. Fabri, E. Nice, 1993, Growth factors.

Overexpression and functional analysis of a mitogen-inducible nuclear GTPase activating protein, Spa-1.

M. Nur-e-Kamal, 1996, The international journal of biochemistry & cell biology.

rho GAP of 28 kDa (GAP2), but not of 190 kDa (p190), requires Asp65 and Asp67 of rho GTPase for its activation.

S. Narumiya, H. Maruta, N. Morii, 1993, The Journal of biological chemistry.

Rsr1 and Rap1 GTPases are activated by the same GTPase-activating protein and require threonine 65 for their activation.

L. Fabri, E. Nice, H. Maruta, 1991, The Journal of biological chemistry.

ras and rap1 GTPases Mutated at Position 64

H. Maruta, M. Nur-e-Kamal, 1991 .

The role of Rho GTPases in the regulation of the rearrangement of actin cytoskeleton and cell movement

R. Begum, M. Nur-E-Kamal, M. Nur-e-Kamal, 2004, Experimental & Molecular Medicine.

Antibacterial activity of Euphorbia thymifolia Linn.

N. Khan, M. Nur-e-Kamal, M. Rahman, 1988, The Indian journal of medical research.

Molecular characterization of erythrocyte glucose-6-phosphate dehydrogenase deficiency in Al-Ain District, United Arab Emirates.

T. Vulliamy, L. Luzzatto, R. Bayoumi, 1996, Human heredity.

Localisation of a gene for an autosomal recessive syndrome of macrocephaly, multiple epiphyseal dysplasia, and distinctive facies to chromosome 15q26

Young-Ae Lee, G. Nürnberg, R. Bayoumi, 2001, Journal of medical genetics.

The CDC42-specific inhibitor derived from ACK-1 blocks v-Ha-Ras-induced transformation

H. Maruta, M. Nur-E-Kamal, M. Nur-e-Kamal, 1999, Oncogene.

Comparative studies on the sensitivity of polymerase chain reaction and microscopic examination for the detection of Trypanosoma evansi in experimentally infected mice.

A. Mohamed, M. K. Ijaz, M. Nur-e-Kamal, 1998, Comparative immunology, microbiology and infectious diseases.

Asparagine 26, glutamic acid 31, valine 45, and tyrosine 64 of Ras proteins are required for their oncogenicity.

A. Sizeland, H. Maruta, G. D'Abaco, 1992, The Journal of biological chemistry.

Homozygosity mapping in families with Joubert syndrome identifies a locus on chromosome 9q34.3 and evidence for genetic heterogeneity.

R. Bayoumi, A. Reis, L. Al-Gazali, 1999, American journal of human genetics.

Molecular cloning of a novel mitogen-inducible nuclear protein with a Ran GTPase-activating domain that affects cell cycle progression

N. Minato, K. Iwai, H. Kubota, 1995, Molecular and cellular biology.

The minimal fragments of c-Raf-1 and NF1 that can suppress v-Ha-Ras-induced malignant phenotype.

H. Maruta, A. Tikoo, M. Fridman, 1994, The Journal of biological chemistry.

The GTPase and Rho GAP domains of p190, a tumor suppressor protein that binds the M(r) 120,000 Ras GAP, independently function as anti-Ras tumor suppressors.

H. Maruta, A. Tikoo, D. Z. Wang, 1997, Cancer research.

Molecular Cloning of the Wild‐Type and Mutant thyA Gene from Shigella flexneri Y

M. Nur-E-Kamal, M. Nur-e-Kamal, Z. Ahmed, 1994, Microbiology and immunology.

Identification of a Neurite Outgrowth-Promoting Motif within the Alternatively Spliced Region of Human Tenascin-C

S. Meiners, M. Nur-E-Kamal, M. Nur-e-Kamal, 2001, The Journal of Neuroscience.

Inhibition of rotavirus infection in vitro and in vivo by a synthetic peptide from VP4.

L. Babiuk, S. Attah-poku, M. K. Ijaz, 1998, Vaccine.

Characterization of a synthetic peptide mimicking trypsin-cleavage site of rotavirus VP4.

L. Babiuk, P. Frenchick, M. K. Ijaz, 1995, Comparative immunology, microbiology and infectious diseases.

Tenascin-C Contains Domains That Independently Regulate Neurite Outgrowth and Neurite Guidance

H. M. Geller, S. Meiners, M. Nur-E-Kamal, 1999, The Journal of Neuroscience.

The GTPase-activating NF1 fragment of 91 amino acids reverses v-Ha-Ras-induced malignant phenotype.

H. Maruta, M. Nur-e-Kamal, M. Varga, 1993, The Journal of biological chemistry.