Bruno Seitaj

Pathophysiological consequences of isoform-specific IP3 receptor mutations.

J. Parys, G. Bultynck, M. Kerkhofs, 2018, Biochimica et biophysica acta. Molecular cell research.

TMBIM5 loss of function alters mitochondrial matrix ion homeostasis and causes a skeletal myopathy

K. Frauenknecht, M. Schmeisser, J. Parys, 2022, Life Science Alliance.

Transmembrane BAX Inhibitor-1 Motif Containing Protein 5 (TMBIM5) Sustains Mitochondrial Structure, Shape, and Function by Impacting the Mitochondrial Protein Synthesis Machinery

S. Tenzer, Ute Distler, J. Parys, 2020, Cells.

Alterations in Ca2+ Signalling via ER-Mitochondria Contact Site Remodelling in Cancer.

P. Pinton, S. Marchi, C. Giorgi, 2017, Advances in experimental medicine and biology.

The trans-membrane domain of Bcl-2α, but not its hydrophobic cleft, is a critical determinant for efficient IP3 receptor inhibition

J. Parys, H. de Smedt, N. Prevarskaya, 2016, Oncotarget.

Ion and pH Sensitivity of a TMBIM Ca2+ Channel.

Lei Shi, M. Quick, Qun Liu, 2019, Structure.

Modulation of Ca2+ Signaling by Anti-apoptotic B-Cell Lymphoma 2 Proteins at the Endoplasmic Reticulum–Mitochondrial Interface

G. Bultynck, G. Monaco, T. Vervliet, 2017, Front. Oncol..