A. Bazarov

发表

PIM kinase inhibition presents a novel targeted therapy against triple-negative breast tumors with elevated MYC expression

Michael T. McManus, Lindsey A. Marsh, C. Yau, 2016 .

Cell Labeling with Magneto-Endosymbionts and the Dissection of the Subcellular Location, Fate, and Host Cell Interactions

B. Rutt, C. Contag, B. Rice, 2018, Molecular Imaging and Biology.

The specific role of pRb in p16INK4A-mediated arrest of normal and malignant human breast cells

S. Lowe, P. Yaswen, A. Chicas, 2012, Cell cycle.

A modest reduction in c-myc expression has minimal effects on cell growth and apoptosis but dramatically reduces susceptibility to Ras and Raf transformation.

J. Sedivy, S. Adachi, M. Mateyak, 2001, Cancer research.

MYC pathway activation in triple-negative breast cancer is synthetic lethal with CDK inhibition

Marc E. Lenburg, Gordon B. Mills, James W. Smyth, 2012, The Journal of experimental medicine.

PIM1 kinase inhibition as a targeted therapy against triple-negative breast tumors with elevated MYC expression

Michael T. McManus, Lindsey A. Marsh, C. Yau, 2016, Nature Medicine.

Inhibition of the Raf-1 kinase by cyclic AMP agonists causes apoptosis of v-abl-transformed cells

W. Kolch, H. Mischak, E. Weissinger, 1997, Molecular and cellular biology.

Formation of higher-order nuclear Rad51 structures is functionally linked to p21 expression and protection from DNA damage-induced apoptosis.

H. Ropers, T. Haaf, J. Sedivy, 2002, Journal of cell science.

Isolation of temperature-sensitive mutations in the c-raf-1 catalytic domain and expression of conditionally active and dominant-defective forms of Raf-1 in cultured mammalian cells.

J. Sedivy, A. Bazarov, K. Lu, 1998, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

Promotion of variant human mammary epithelial cell outgrowth by ionizing radiation: an agent-based model supported by in vitro studies

Sylvain V Costes, Mary Helen Barcellos-Hoff, Rituparna Mukhopadhyay, 2010, Breast Cancer Research.

p16INK4a‐mediated suppression of telomerase in normal and malignant human breast cells

S. Lowe, B. Ren, Leonard K. Lee, 2010, Aging cell.

Telomerase activation by c-Myc in human mammary epithelial cells requires additional genomic changes

A. Béliveau, P. Yaswen, W. C. Hines, 2009, Cell cycle.

Title A tumor-suppressing function in the epithelial adhesion protein

Stephen, Donghui Wang, B. Hann, 2016 .

A tumor suppressing function in the epithelial adhesion protein Trask

Donghui Wang, B. Hann, M. Moasser, 2011, Oncogene.

BORIS (CTCFL) Is Not Expressed in Most Human Breast Cell Lines and High Grade Breast Carcinomas

P. Yaswen, W. C. Hines, R. Mukhopadhyay, 2010, PloS one.

Who is in the driver's seat in 8p12 amplifications? ZNF703 in luminal B breast tumors

P. Yaswen, A. Bazarov, 2011, Breast Cancer Research.

A Simple Three-dimensional Hydrogel Platform Enables Ex Vivo Cell Culture of Patient and PDX Tumors for Assaying Their Response to Clinically Relevant Therapies

K. Hribar, C. Patil, C. Wheeler, 2019, Molecular Cancer Therapeutics.

Lawrence Berkeley National Laboratory Recent Work Title Who is in the driver ' s seat in 8 p 12 amplifications ? ZNF 703 in luminal B breast tumors

P. Yaswen, A. Bazarov, 2022 .

PIM1 kinase inhibition as a targeted therapy against triple-negative breast tumors with elevated MYC expression

Michael T. McManus, Lindsey A. Marsh, C. Yau, 2016, Nature Medicine.

The specific role of pRb in p16INK4A-mediated arrest of normal and malignant human breast cells

S. Lowe, P. Yaswen, A. Chicas, 2012, Cell cycle.