James P. Barrett

发表

Bone marrow-derived macrophages from aged rats are more responsive to inflammatory stimuli

M. Lynch, James P. Barrett, Derek A Costello, 2015, Journal of Neuroinflammation.

Bidirectional Brain-Systemic Interactions and Outcomes After TBI

B. Stoica, A. Faden, R. Henry, 2021, Trends in Neurosciences.

Bone marrow-derived macrophages from AβPP/PS1 mice are sensitized to the effects of inflammatory stimuli.

M. Lynch, Raasay S. Jones, R. Kelly, 2015, Journal of Alzheimer's disease : JAD.

NOX2 deficiency alters macrophage phenotype through an IL-10/STAT3 dependent mechanism: implications for traumatic brain injury

B. Stoica, A. Faden, S. Villapol, 2017, Journal of Neuroinflammation.

NOX2 drives M1-like microglial/macrophage activation and neurodegeneration following experimental traumatic brain injury

B. Stoica, A. Faden, D. Loane, 2016, Brain, Behavior, and Immunity.

Down-Regulation of miR-23a-3p Mediates Irradiation-Induced Neuronal Apoptosis

B. Stoica, A. Faden, B. Sabirzhanov, 2020, International journal of molecular sciences.

Novel mGluR5 Positive Allosteric Modulator Improves Functional Recovery, Attenuates Neurodegeneration, and Alters Microglial Polarization after Experimental Traumatic Brain Injury

B. Stoica, A. Faden, F. Tchantchou, 2014, Neurotherapeutics.

LPS-induced release of IL-6 from glia modulates production of IL-1β in a JAK2-dependent manner

M. Lynch, A. Minogue, James P. Barrett, 2012, Journal of Neuroinflammation.

Inhibition of miR-155 Limits Neuroinflammation and Improves Functional Recovery After Experimental Traumatic Brain Injury in Mice

B. Stoica, A. Faden, D. Loane, 2018, Neurotherapeutics.

Early or Late Bacterial Lung Infection Increases Mortality After Traumatic Brain Injury in Male Mice and Chronically Impairs Monocyte Innate Immune Function

A. Faden, S. Vogel, D. Loane, 2020, Critical care medicine.

Involvement of IGF-1 and Akt in M1/M2 activation state in bone marrow-derived macrophages.

M. Lynch, A. Minogue, James P. Barrett, 2015, Experimental cell research.

Inflammatory response of microglia to prions is controlled by sialylation of PrPSc

N. Makarava, I. Baskakov, D. Loane, 2018, Scientific Reports.

Chronic Alterations in Systemic Immune Function after Traumatic Brain Injury.

A. Faden, Elise L. Ma, D. Loane, 2018, Journal of neurotrauma.

Microglial Depletion with CSF1R Inhibitor During Chronic Phase of Experimental Traumatic Brain Injury Reduces Neurodegeneration and Neurological Deficits

B. Stoica, A. Faden, Y. Jiao, 2019, The Journal of Neuroscience.

Inhibition of miR-155 Limits Neuroinflammation and Improves Functional Recovery After Experimental Traumatic Brain Injury in Mice

B. Stoica, A. Faden, D. Loane, 2018, Neurotherapeutics.

LPS-induced release of IL-6 from glia modulates production of IL-1β in a JAK2-dependent manner

M. Lynch, A. Minogue, James P. Barrett, 2012, Journal of Neuroinflammation.

Irradiation-Induced Upregulation of miR-711 Inhibits DNA Repair and Promotes Neurodegeneration Pathways

B. Stoica, A. Faden, B. Sabirzhanov, 2020, International journal of molecular sciences.

Bone marrow-derived macrophages from aged rats are more responsive to inflammatory stimuli

M. Lynch, James P. Barrett, T. Cowley, 2015, Journal of Neuroinflammation.

NOX2 deficiency alters macrophage phenotype through an IL-10/STAT3 dependent mechanism: implications for traumatic brain injury

B. Stoica, A. Faden, S. Villapol, 2017, Journal of Neuroinflammation.

Interferon-β Plays a Detrimental Role in Experimental Traumatic Brain Injury by Enhancing Neuroinflammation That Drives Chronic Neurodegeneration

B. Stoica, A. Faden, S. Vogel, 2020, The Journal of Neuroscience.

Traumatic Brain Injury Induces cGAS Activation and Type I Interferon Signaling in Aged Mice

B. Stoica, Surajit Bhattacharya, D. Loane, 2021, Frontiers in Immunology.

Inflammatory response of microglia to prions is controlled by sialylation of PrPSc

N. Makarava, I. Baskakov, D. Loane, 2018, Scientific Reports.

Novel mGluR5 Positive Allosteric Modulator Improves Functional Recovery, Attenuates Neurodegeneration, and Alters Microglial Polarization after Experimental Traumatic Brain Injury

B. Stoica, A. Faden, F. Tchantchou, 2014, Neurotherapeutics.

Interaction of high-fat diet and brain trauma alters adipose tissue macrophages and brain microglia associated with exacerbated cognitive dysfunction

B. Stoica, A. Faden, R. Henry, 2023, bioRxiv.

Interaction of high-fat diet and brain trauma alters adipose tissue macrophages and brain microglia associated with exacerbated cognitive dysfunction

R. Henry, James P. Barrett, Oleg Makarevich, 2024, Journal of neuroinflammation.