Shidong Ma

发表

PD-1/CTLA-4 Blockade Inhibits Epstein-Barr Virus-Induced Lymphoma Growth in a Cord Blood Humanized-Mouse Model

S. Kenney, Richard J. Jones, J. Gumperz, 2016, PLoS pathogens.

The Epstein‐Barr virus EBNA2 protein induces a subset of NOTCH target genes in thyroid cancer cell lines but fails to suppress proliferation

Herb Chen, S. Kenney, A. Marín, 2017, Surgery.

Human NKT cells direct the differentiation of myeloid APCs that regulate T cell responses via expression of programmed cell death ligands.

S. Kenney, J. Gumperz, Subramanya Hegde, 2011, Journal of autoimmunity.

Leflunomide/teriflunomide inhibit Epstein-Barr virus (EBV)-induced lymphoproliferative disease and lytic viral replication

S. Kenney, Zhe Li, M. Tsai, 2017, Oncotarget.

LMP1-deficient Epstein-Barr virus mutant requires T cells for lymphomagenesis.

M. Gulley, E. Cesarman, E. Ranheim, 2015, The Journal of clinical investigation.

A New Model of Epstein-Barr Virus Infection Reveals an Important Role for Early Lytic Viral Protein Expression in the Development of Lymphomas

M. Gulley, K. Young, S. Kenney, 2010, Journal of Virology.

Mice engrafted with human fetal thymic tissue and hematopoietic stem cells develop pathology resembling chronic graft-versus-host disease.

S. Kenney, W. Burlingham, P. Hematti, 2013, Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation.

5-hydroxymethylation of the EBV genome regulates the latent to lytic switch

E. Johannsen, S. Kenney, P. Lambert, 2015, Proceedings of the National Academy of Sciences.

An Epstein-Barr Virus (EBV) Mutant with Enhanced BZLF1 Expression Causes Lymphomas with Abortive Lytic EBV Infection in a Humanized Mouse Model

M. Gulley, J. Mertz, S. Kenney, 2012, Journal of Virology.

Latent Membrane Protein 1 (LMP1) and LMP2A Collaborate To Promote Epstein-Barr Virus-Induced B Cell Lymphomas in a Cord Blood-Humanized Mouse Model but Are Not Essential

E. Ranheim, S. Kenney, M. Tsai, 2017, Journal of Virology.

Adoptively transferred Vγ9Vδ2 T cells show potent antitumor effects in a preclinical B cell lymphomagenesis model.

S. Kenney, J. Gumperz, C. L. Schneider, 2017, JCI insight.

Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus’s natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoter

E. Johannsen, R. Kraus, J. Mertz, 2017, PLoS pathogens.

An EBNA3A-Mutated Epstein-Barr Virus Retains the Capacity for Lymphomagenesis in a Cord Blood-Humanized Mouse Model

E. Johannsen, E. Ranheim, S. Kenney, 2020, Journal of Virology.

An EBNA3C-deleted Epstein-Barr virus (EBV) mutant causes B-cell lymphomas with delayed onset in a cord blood-humanized mouse model

E. Johannsen, E. Ranheim, S. Kenney, 2018, PLoS pathogens.

Hsp90 inhibitors block outgrowth of EBV-infected malignant cells in vitro and in vivo through an EBNA1-dependent mechanism

R. Burgess, S. Ma, R. Khanna, 2010, Proceedings of the National Academy of Sciences.

Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial Cells

E. Johannsen, J. Mertz, S. Kenney, 2015, PLoS pathogens.

Mice engrafted with human fetal thymic tissue and hematopoietic stem cells develop pathology resembling chronic graft-versus-host disease.

S. Kenney, W. Burlingham, P. Hematti, 2013, Biology of blood and marrow transplantation : journal of the American Society for Blood and Marrow Transplantation.